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As a group, atrial septal defects are detected in one child per 1500 live births. PFOs are quite common (appearing in 10–20% of adults), but when asymptomatic go undiagnosed. ASDs make up 30 to 40% of all congenital heart diseases that are seen in adults. [58] The ostium secundum atrial septal defect accounts for 7% of all congenital heart ...
[5] The development of pulmonary hypertension is very serious. And this because the left ventricle is weakened due to its overuse. When this happens, the pressure backs up into the pulmonary veins and the lungs. [5] This type of damage is irreversible which is why immediate treatment is recommended after diagnosis. [6]
The septum primum is on the left side of the heart in the left atrium while the septum secundum is much thicker and is located on the right side, in the right atrium. During development, blood shunts from the floor of the right atrium through the foramen ovale in the septum secundum then up through the foramen secundum in the septum primum. [2]
This is a very serious condition and surgery is necessary within the first six months of life for a child. [2] Half of the children who are untreated with this condition die during their first year due to heart failure or pneumonia. [3] Atrioventricular canal defect is a combination of abnormalities of the heart and is present at birth.
Congenital heart defects are divided into two main groups: cyanotic heart defects and non-cyanotic heart defects, depending on whether the child has the potential to turn bluish in color. [3] The defects may involve the interior walls of the heart, the heart valves , or the large blood vessels that lead to and from the heart.
Cardiac activity is visible beginning at approximately 5 weeks of pregnancy. The human heart begins beating at a rate near the mother's, about 75-80 beats per minute (BPM). The embryonic heart rate (EHR) then accelerates linearly for the first month of beating, peaking at 165-185 BPM during the early 7th week, (early 9th week after the LMP).
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Congenital heart disease, particularly VSDs, is the number one cause of death for children with Down syndrome ages birth to two. [7] A VSD can also form a few days after a myocardial infarction [8] (heart attack) due to mechanical tearing of the septal wall, before scar tissue forms, when macrophages start remodeling the dead heart tissue.