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Cheyne–Stokes respiration is a breathing pattern consisting of alternating periods of rapid and slow breathing, which may result from a brain stem injury. [12] Cheyne-Stokes respiration may be observed in newborn babies, but this is occasionally physiological (normal). Chest retractions may be observed in patients with asthma.
Labored breathing is distinguished from shortness of breath or dyspnea, which is the sensation of respiratory distress rather than a physical presentation.. Still, many [2] simply define dyspnea as difficulty in breathing without further specification, which may confuse it with e.g. labored breathing or tachypnea (rapid breathing). [3]
Tachypnea, also spelt tachypnoea, is a respiratory rate greater than normal, resulting in abnormally rapid and shallow breathing. [1]In adult humans at rest, any respiratory rate of 12–20 per minute is considered clinically normal, with tachypnea being any rate above that. [2]
increased work of breathing; nasal flaring (nostrils widening during breathing) accessory muscle use (using muscles other than the diaphragm and intercostal muscles during breathing (e.g., sternocleidomastoid)) retractions (collapse of parts of the chest during breathing) (e.g., suprasternal, substernal, sternal, intercostal, subcostal ...
The diaphragm is the major muscle responsible for breathing.It is a thin, dome-shaped muscle that separates the abdominal cavity from the thoracic cavity. During inhalation, the diaphragm contracts, so that its center moves caudally (downward) and its edges move cranially (upward).
Eupnea is an efficient and effective form of breathing, which balances between maximizing air intake, and minimizing muscular effort. During eupnea, neural output to respiratory muscles is highly regular and stable, with rhythmic bursts of activity during inspiration only to the diaphragm and external intercostal muscles.
Causes may include heart failure, kidney failure, narcotic poisoning, intracranial pressure, and hypoperfusion of the brain (particularly of the respiratory center). The pathophysiology of Cheyne–Stokes breathing can be summarized as apnea leading to increased CO 2 which causes excessive compensatory hyperventilation, in turn causing decreased CO 2 which causes apnea, restarting the cycle.
Breathing is normally an unconscious, involuntary, automatic process. The pattern of motor stimuli during breathing can be divided into an inhalation stage and an exhalation stage. Inhalation shows a sudden, ramped increase in motor discharge to the respiratory muscles (and the pharyngeal constrictor muscles ). [ 5 ]