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Sarcoidosis is a disease of unknown cause characterized by non-necrotizing ("non-caseating") granulomas in multiple organs and body sites, [12] most commonly the lungs and lymph nodes within the chest cavity. Other common sites of involvement include the liver, spleen, skin, and eyes.
The World Association of Sarcoidosis and Other Granulomatous Disorders (WASOG) is an organisation of physicians involved in the diagnosis and treatment of sarcoidosis and related conditions. [171] WASOG publishes the journal Sarcoidosis, Vasculitis and Diffuse Lung Diseases. [172]
On rare occasions, thoracoscopic lung biopsy is required. On histopathological examination, a biopsy will show leukocytoclastic vasculitis with necrotic changes and granulomatous inflammation (clumps of typically arranged white blood cells) on microscopy. These granulomas are the main reason for the name granulomatosis with polyangiitis ...
Focal necrotizing vasculitis, necrotizing granulomas in the lung and upper airway, necrotizing glomerulonephritis: Granulomatosis with polyangiitis: Hakim Triad [1] (Adams Triad) Urinary incontinence, Gait disturbance, Dementia: Normal pressure hydrocephalus: Triad (Triad of Sandblom) Malena, Obstructive jaundice, Biliary colic: Hemobilia ...
Standard bronchoscopy usually shows chronic inflammation of lung tissue, and granulomas with poorly-defined margins. [1] Lung biopsy usually reveals non-necrotizing granulomatous inflammation. [7] It also shows "foamy" macrophages and crystals of oxalic acid. [3] Hyperplasia tends to occur in type 1 pneumocytes and not in type 2 pneumocytes. [3]
Granuloma formation is seen in other diseases such as tuberculosis and sarcoidosis. Sarcoidosis, like berylliosis, also produces granulomas without a necrotic core, and so distinction between the two processes can be difficult. Ultimately, this process leads to restrictive lung disease (a decrease in diffusion capacity).
Chronic pulmonary aspergillosis is a long-term fungal infection caused by members of the genus Aspergillus—most commonly Aspergillus fumigatus. [8] The term describes several disease presentations with considerable overlap, ranging from an aspergilloma [12] —a clump of Aspergillus mold in the lungs—through to a subacute, invasive form known as chronic necrotizing pulmonary aspergillosis ...
Caseous necrosis in the kidney. In caseous necrosis no histological architecture is preserved (unlike with coagulative necrosis). [5] [6] On microscopic examination with H&E staining, the area is acellular, characterised by amorphous, roughly granular eosinophilic debris of now dead cells, [6] also containing interspearsed haematoxyphilic remnants of cell nucleus contents. [5]
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