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  2. Congenital adrenal hyperplasia due to 17α-hydroxylase deficiency

    en.wikipedia.org/wiki/Congenital_adrenal...

    [citation needed] In women with mild cases, elevated blood pressure and/or infertility is the presenting clinical problem. 17α-hydroxylase deficiency in genetic males results in moderate to severe reduction of fetal testosterone production by adrenal glands and testes. Undervirilization is variable and sometimes complete.

  3. 5α-Reductase 2 deficiency - Wikipedia

    en.wikipedia.org/wiki/5α-Reductase_2_deficiency

    A high level of assay specificity is required to measure concentrations of DHT since serum T levels are generally 10-fold higher than DHT in young males. Endocrinological tests for T/DHT ratios can be difficult to interpret since the normal ratio level varies according to age and severity of 5αR2 activity impairment. Affected young males of at ...

  4. Congenital adrenal hyperplasia - Wikipedia

    en.wikipedia.org/wiki/Congenital_adrenal_hyperplasia

    Currently, in the United States and over 40 other countries, every child born is screened for 21-hydroxylase CAH at birth. This test detects elevated levels of 17α-hydroxyprogesterone (17-OHP). Detecting high levels of 17-OHP enables early detection of CAH. Newborns detected early enough can be placed on medication and live relatively normal ...

  5. Late onset congenital adrenal hyperplasia - Wikipedia

    en.wikipedia.org/wiki/Late_onset_congenital...

    The condition of 21-hydroxylase deficiency is screened by measuring serum levels of 17α-hydroxyprogesterone (17-OHP) in the morning and between day 3 and 5 of the menstrual cycle (for females) to reduce the possibility of false positive results. [12] 17-OHP is used as a marker of the 21-hydroxylase enzyme activity since the 1980s. [52]

  6. Congenital adrenal hyperplasia due to 21-hydroxylase deficiency

    en.wikipedia.org/wiki/Congenital_adrenal...

    A study has revealed that serum progesterone concentrations in boys (10 days to 18 years old) with 21-hydroxylase deficiency reached levels up to 10.14 ng/mL, i.e. similar to female luteal values, while in the control group of boys, average level was 0.07 ng/mL (0.22 nmol/L), with values ranging from 0.05 to 0.40 ng/mL. [38]

  7. 17β-Hydroxysteroid dehydrogenase III deficiency - Wikipedia

    en.wikipedia.org/wiki/17β-Hydroxysteroid...

    17-β-Hydroxysteroid dehydrogenase III deficiency is a cause of 46,XY disorder of sex development (46,XY DSD) that presents in males with variable effects on genitalia which can be complete or predominantly female with a blind vaginal pouch. Testes are often found in the inguinal canal or in a bifid scrotum.

  8. Congenital adrenal hyperplasia due to 3β-hydroxysteroid ...

    en.wikipedia.org/wiki/Congenital_adrenal...

    The issues surrounding corrective surgery of the virilized female genitalia are the same as for moderate 21-hydroxylase deficiency but surgery is rarely considered desirable. [ citation needed ] The extent to which mild 3β-HSD CAH can cause early appearance of pubic hair and other aspects of hyperandrogenism in later childhood or adolescence ...

  9. Isolated 17,20-lyase deficiency - Wikipedia

    en.wikipedia.org/.../Isolated_17,20-lyase_deficiency

    The symptoms of isolated 17,20-lyase deficiency, in males, include pseudohermaphroditism (i.e., feminized, ambiguous, or mildly underdeveloped (e.g., micropenis, perineal hypospadias, and/or cryptorchidism (undescended testes)) external genitalia), female gender identity, and, in non-complete cases of deficiency where partial virilization occurs, gynecomastia up to Tanner stage V (due to low ...

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