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Hepatomegaly is enlargement of the liver. [4] It is a non-specific medical sign , having many causes, which can broadly be broken down into infection , hepatic tumours , and metabolic disorder . Often, hepatomegaly presents as an abdominal mass .
Hepatomegaly Medical imaging : Longitudinal axis > 15.5 cm at the hepatic midline, or > 16.0 cm at the midclavicular line [ 14 ] Autopsy : Weight over upper limit of standard reference range, of 1,860 g (4.10 lb) in men [ 12 ] and 1,770 g (3.90 lb) in women.
The liver span is a measurement performed during physical examination to determine the size of the liver and identify possible hepatomegaly.. It is the distance between the lower border of the liver in the mid-clavicular line obtained by palpation, and the upper border of the liver in the mid-clavicular line detected by percussion (the upper border of the liver lies behind the ribs and can not ...
Tachycardia, Tachypnea, Tender hepatomegaly: Congestive heart failure: Cushing's triad of Increased Intracranial Pressure: Bradycardia, Bradypnea, Hypertension: Head injuries Danny's Triad: Painful urination, testicular pain, diarrhea: Chlamydia Dieulafoy's triad: hyperesthesia of the skin, exquisite tenderness and guarding over McBurney's ...
Fatty liver disease (FLD), also known as hepatic steatosis and steatotic liver disease (SLD), is a condition where excess fat builds up in the liver. [1] Often there are no or few symptoms.
Hepatomegaly with enlarged liver visible in red crosshairs and extending downward. Impairment in the liver's ability to perform gluconeogenesis leads to clinically apparent hepatomegaly . Without this process, the body is unable to liberate glycogen from the liver and convert it into blood glucose, leading to an accumulation of stored glycogen ...
Inflammatory cytokines (TNF-alpha, IL-6 and IL-8) are thought to be essential in the initiation and perpetuation of liver injury and cytotoxic hepatomegaly by inducing apoptosis and severe hepatotoxicity. One possible mechanism for the increased activity of TNF-α is the increased intestinal permeability due to liver disease.
Patients with Mauriac syndrome may present with obesity, hepatomegaly, cushingoid facies, and elevated liver enzymes. Patients usually have growth failure and delayed puberty, which should warn the physician about insufficient management of the patient's diabetes. These symptoms can be reversed with good glycemic control. [3] [4] [5]