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Gestational diabetes can develop in dogs as well. It can be prevented by behavioral and dietary management. [13] Diabetes insipidus, which has nothing to do with blood sugar, but is a condition of insufficient antidiuretic hormone or resistance to it, also exists in dogs. [14] [15]
For dogs under a year there are Puppy Growth Charts [9] where weights can be monitored in relation to an average growth curve, this again helps to monitor whether weight is increasing more than expected and deviating from the average growth curve should prompt assessing BCS.
The first dog trained to detect hypoglycemia was a Californian dog called Armstrong in 2003. [5] In 2009, a dog named Tinker from Durham City became the first self-taught British assistance dog to be officially registered for a type 2 diabetic owner. He was able to give his owner Paul Jackson up to half an hour warning before an attack occurred ...
The fluctuation of blood sugar (red) and the sugar-lowering hormone insulin (blue) in humans during the course of a day with three meals. One of the effects of a sugar-rich vs a starch-rich meal is highlighted. [1] The blood sugar level, blood sugar concentration, blood glucose level, or glycemia is the measure of glucose concentrated in the blood.
The glucose tolerance test was first described in 1923 by Jerome W. Conn. [4]The test was based on the previous work in 1913 by A. T. B. Jacobson in determining that carbohydrate ingestion results in blood glucose fluctuations, [5] and the premise (named the Staub-Traugott Phenomenon after its first observers H. Staub in 1921 and K. Traugott in 1922) that a normal patient fed glucose will ...
Graph depicting blood sugar change during a day with three meals. The glycemic (glycaemic) index (GI; / ɡ l aɪ ˈ s iː m ɪ k / [1]) is a number from 0 to 100 assigned to a food, with pure glucose arbitrarily given the value of 100, which represents the relative rise in the blood glucose level two hours after consuming that food. [2]
Splay is usually used in reference to glucose; [1] other substances, such as phosphate, have virtually no splay at all. The splay in the glucose titration curve is likely a result of both anatomical and kinetic difference among nephrons. [7] A particular nephron's filtered load of glucose may be mismatched to its capacity to reabsorb glucose.
The AGP collapses the two weeks of glucose data and plots only by time allowing for underlying patterns to be identified. It uses five smoothed frequency curves to represent glucose exposure, variability and stability while simultaneously identifying periods of significant hypoglycemia and hyperglycemia. [11]
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