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Reproductive toxicants may adversely affect sexual function, ovarian failure, fertility as well as causing developmental toxicity in the offspring. [2] [3] Lowered effective fertility related to reproductive toxicity relates to both male and female effects alike and is reflected in decreased sperm counts, semen quality and ovarian failure.
Reproductive toxins (3 C, 1 P) A. Abortifacients (42 P) Pages in category "Reproductive toxicants" The following 2 pages are in this category, out of 2 total.
Pages in category "Reproductive toxins" This category contains only the following page. This list may not reflect recent changes. C. ContraPest
By contrast, a toxin is a poison produced naturally by an organism (e.g. plant, animal, insect). [2] The 2011 book A Textbook of Modern Toxicology states, "A toxin is a toxicant that is produced by a living organism and is not used as a synonym for toxicant—all toxins are toxicants, but not all toxicants are toxins.
A toxin is a naturally occurring poison [1] produced by metabolic activities of living cells or organisms. [2] They occur especially as proteins , often conjugated . [ 3 ] The term was first used by organic chemist Ludwig Brieger (1849–1919), [ 4 ] derived from toxic .
Shigella dysenteriae, which produces dysentery has toxins that fall into two major groups, Stx1 and Stx2, whose genes are considered to be part of the genome of lambdoid prophages. Streptococcus pyogenes , produce a pyrogenic exotoxin , obtained by lysogenic conversion, which causes fever and a scarlet-red rash, scarlet fever .
By tissue target type susceptible to the toxin (neurotoxins affect the nervous system, cardiotoxins affect the heart, etc.) By structure (for example, AB5 toxin) By domain architecture of the toxin (for example, polymorphic toxins) By the ability of the toxin to endure in hostile environments, such as heat, dryness, radiation, or salinity.
SpeB was identified in 1919 as an ectoenzyme secreted by certain strains of streptococci. [11] It was originally studied as two separate toxins, streptococcal pyrogenic exotoxin B and streptococcal cysteine proteinase, until it was shown that both proteins were encoded by the speB gene and that the attributed pyrogenic activities were due to contamination by SpeA and SpeC.