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The treatment of takotsubo cardiomyopathy is generally supportive in nature, for it is considered a transient disorder. [45] Treatment is dependent on whether patients experience heart failure or acute hypotension and shock. In many individuals, left ventricular function normalizes within two months.
In radial section, two tracheids of a coniferous wood species are shown. A series of bordered pits are also appearing in each tracheid. A tracheid of oak shows pits along the walls. It has no perforation plates. Angiosperms have both tracheids and vessel elements. [1] A tracheid is a long and tapered lignified cell in the xylem of vascular ...
A heart attack occurs suddenly when an atherosclerotic plaque in one of the arteries to your heart ruptures. It can cause symptoms such as: Chest pain, pressure, or tightness
Ischemic cardiomyopathy is a type of cardiomyopathy caused by a narrowing of the coronary arteries which supply blood to the heart. [4] Typically, patients with ischemic cardiomyopathy have a history of acute myocardial infarction, [5] however, it may occur in patients with coronary artery disease, but without a past history of acute myocardial infarction.
Heart disease is the number one killer of men and women in the United States—it kills more people than all cancers combined, and is responsible for one out of every four deaths, according to the ...
A heart attack can cause arrhythmias, as well as permanent damage to the heart muscle. [25] Coronary ischemia resulting from coronary artery disease also increases the risk of developing heart failure. [10] Most cases of heart failure result from underlying coronary artery disease. [10]
Coronary thrombosis is defined as the formation of a blood clot inside a blood vessel of the heart. This blood clot may then restrict blood flow within the heart, leading to heart tissue damage, or a myocardial infarction, also known as a heart attack.
An increase in sympathetic stimulation to the heart increases contractility and heart rate. An increase in contractility tends to increase stroke volume and thus a secondary increase in preload. An increase in preload results in an increased force of contraction by Starling's law of the heart; this does not require a change in contractility.
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