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Brugada syndrome is diagnosed by identifying characteristic patterns on an electrocardiogram. [14] The pattern seen on the ECG includes ST elevation in leads V 1-V 3 with a right bundle branch block (RBBB) appearance. There may be evidence of a slowing of electrical conduction within the heart, as shown by a prolonged PR interval.
Rare diseases called ion channelopathies may play a role such as long QT syndrome (LQTS), Brugada syndrome (BrS), CPVT (catecholaminergic polymorphic ventricular tachycardia), progressive cardiac conduction defect (PCCD), early repolarization syndrome, mixed sodium channel disease, and short QT syndrome. [13]
Sudden unexplained nocturnal death syndrome may refer to: Brugada syndrome , a genetic disorder in which the electrical activity within the heart is abnormal Sudden arrhythmic death syndrome (SADS), a sudden unexpected death of adolescents and adults, mainly during sleep
Sodium channel, implicated in channelopathies including Brugada syndrome, Long QT syndrome, Dravet syndrome, Paramyotonia congenita: ... Due to similar symptoms ...
The resting 12-lead ECG is a useful test to differentiate CPVT from other electrical diseases of the heart that can cause similar abnormal heart rhythms. Unlike conditions such as long QT syndrome and Brugada syndrome, the resting 12-lead ECG in those with CPVT is generally normal. [8]
Brugada syndrome is a genetic disease that can result in mutations in the sodium ion channel (gene SCN5A) of the myocytes in the heart. [10] Brugada syndrome can result in ventricular fibrillation and potentially death. It is a major cause of sudden unexpected cardiac death in young, otherwise healthy people. [11]
Brown–Vialetto–Van Laere syndrome; Bruck syndrome; Brugada syndrome; Brunner syndrome; Budd–Chiari syndrome; Burning feet syndrome; Burning mouth syndrome; Burnside–Butler syndrome; Buschke–Ollendorff syndrome; Bálint's syndrome; Börjeson-Forssman-Lehmann syndrome
Long QT syndrome is estimated to affect 1 in 7,000 people. [6] Females are affected more often than males. [6] Most people with the condition develop symptoms before they are 40 years old. [6] It is a relatively common cause of sudden death along with Brugada syndrome and arrhythmogenic right ventricular dysplasia. [3]