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Frontal bossing is the development of an unusually pronounced forehead which may also be associated with a heavier than normal brow ridge. It is caused by enlargement of the frontal bone , often in conjunction with abnormal enlargement of other facial bones , skull , mandible , and bones of the hands and feet.
Parrot's sign (19th century), refers to at least two medical signs; one relating to a large skull and another to a pupil reaction. [1]One Parrot's sign describes the bony growth noted at autopsy by Joseph-Marie-Jules Parrot [] and Jonathan Hutchinson on the skulls of children with congenital syphilis (CS) in the 19th century.
Acute periostitis is due to infection, characterized by diffuse formation of pus, severe pain, and constitutional symptoms, and usually results in necrosis.It can be caused by excessive physical activity as well, as in the case of medial tibial stress syndrome (also referred to as tibial periostalgia, soleus periostalgia, or shin splints).
Compensatory growth occurs forward at the coronal suture and backward at the lambdoid suture giving respectively a prominent forehead, called frontal bossing, and a prominent back portion of the head, called coning. [10] [11] This is the most common form of craniosynostosis. [13]
Paget's disease affecting the skull may cause frontal bossing, increased hat size, and headaches. Often patients may develop loss of hearing in one or both ears [ 7 ] due to auditory foramen narrowing and resultant compression of the nerves in the inner ear.
High anterior hairline is a dysmorphic feature in which the frontal hairline which defines the top and sides of the forehead is unusually high. This can mean that either the distance between the trichion (hairline) and glabella is more than 2 SD above the mean, or that this distance is apparently (subjectively) increased.
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The disease was first described by Maroteaux and Lamy in 1962 [4] [5] at which time it was defined by the following characteristics: dwarfism; osteopetrosis; partial agenesis of the terminal digits of the hands and feet; cranial anomalies, such as persistence of fontanelles and failure of closure of cranial sutures; frontal and occipital bossing; and hypoplasia of the angle of the mandible. [6]