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Women are far more likely than men to get autoimmune diseases, when an out-of-whack immune system attacks their own bodies — and new research may finally explain why. It’s all about how the ...
Men have higher mortality. [8] SLE significantly increases the risk of cardiovascular disease, with this being the most common cause of death. [5] While women with lupus have higher risk pregnancies, most are successful. [1] Rate of SLE varies between countries from 20 to 70 per 100,000. [2] Young women are affected about nine times more often ...
It could make cancer more deadly, study says ... “The fundamental new knowledge we provide here may explain why certain cancers are worse in either men or women, and how best to treat them ...
More than 24 million Americans, by some estimates up to 50 million, have an autoimmune disorder — diseases such as lupus, rheumatoid arthritis, multiple sclerosis and dozens more. About 4 of every 5 patients are women, a mystery that has baffled scientists for decades. One theory is that the X chromosome might be a culprit.
Researchers at Northwestern Medicine and Brigham and Women’s Hospital say they’ve discovered a root cause of lupus, a disease that affects hundreds of thousands of people in the U.S.
Lupus erythematosus is a collection of autoimmune diseases in which the human immune system becomes hyperactive and attacks healthy tissues. [1] Symptoms of these diseases can affect many different body systems, including joints, skin, kidneys, blood cells, heart, and lungs.
Discoid lupus erythematosus is the most common type of chronic cutaneous lupus (CCLE), an autoimmune skin condition on the lupus erythematosus spectrum of illnesses. [ 1 ] [ 2 ] It presents with red, painful, inflamed and coin-shaped patches of skin with a scaly and crusty appearance, most often on the scalp, cheeks, and ears.
Tumid lupus erythematosus is a rare, but distinctive entity in which patients present with edematous erythematous plaque. [ 2 ] Lupus erythematosus tumidus (LET) was reported by Henri Gougerot and Burnier R. in 1930.