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Stomach pain is a common symptom of functional dyspepsia. Symptoms: Early satiety, heartburn, nausea, postprandial fullness, vomiting, and/or epigastric pain. [1] Complications: Symptoms of anxiety, depression, and somatization. [2] Types: Postprandial distress syndrome and epigastric pain syndrome. [1] Diagnostic method: Rome IV criteria. [1 ...
Abdominal angina usually starts 30 minutes after eating and persists for one to three hours. Individuals typically express the pain as a dull ache by clenching their fists over the epigastrium (Levine sign). [3] Sometimes people may reduce their caloric intake in an attempt to decrease pain which can lead to weight loss.
Superior mesenteric artery compressing the duodenum, featuring the superior mesenteric artery syndrome. Superior mesenteric artery (SMA) syndrome is a gastro-vascular disorder in which the third and final portion of the duodenum is compressed between the abdominal aorta (AA) and the overlying superior mesenteric artery.
Lower left abdominal pain can have many causes, ranging from minor to serious, says Andrew Boxer, M.D., gastroenterologist of Gastroenterology Associates of New Jersey. Common causes include ...
The chronic form typically presents more gradually with abdominal pain after eating, unintentional weight loss, vomiting, and fear of eating. [ 1 ] [ 2 ] Risk factors for acute intestinal ischemia include atrial fibrillation , heart failure , chronic kidney failure , being prone to forming blood clots , and previous myocardial infarction . [ 2 ]
The most common symptoms of a duodenal ulcer are waking at night with upper abdominal pain, and upper abdominal pain that improves with eating. [1] With a gastric ulcer, the pain may worsen with eating. [8] The pain is often described as a burning or dull ache. [1] Other symptoms include belching, vomiting, weight loss, or poor appetite. [1]
An analysis by University Hospitals Cleveland Medical Center of records from the TriNetX database found that the number of patients diagnosed with gastroparesis after being prescribed a GLP-1 receptor agonist (0.1% of the patients) was 250% greater than the number of patients diagnosed with gastroparesis who did not take a GLP-1 medication (0.04%).
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