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Abnormal Q waves — 31 percent sensitivity; Cabrera's sign — 27 percent sensitivity, 47 percent for anteroseptal MI; Initial positivity in V1 with a Q wave in V6 — 20 percent sensitivity but 100 percent specificity for anteroseptal MI
In lead V 1, the QRS complex is often entirely negative (QS morphology), although a small initial R wave may be seen (rS morphology). In the lateral leads (I, aVL, V 5-V 6) the QRS complexes are usually predominantly positive with a slow upstroke last >60ms to the R-wave peak. [4] Notching may be seen in these leads but this is not universal.
Hyperacute T waves need to be distinguished from the peaked T waves associated with hyperkalemia. [16] In the first few hours the ST segments usually begin to rise. [17] Pathological Q waves may appear within hours or may take greater than 24 hr. [17] The T wave will generally become inverted in the first 24 hours, as the ST elevation begins to ...
The T wave represents the repolarization of the ventricles. It is generally upright in all leads except aVR and lead V1. Inverted T waves can be a sign of myocardial ischemia, left ventricular hypertrophy, high intracranial pressure, or metabolic abnormalities. Peaked T waves can be a sign of hyperkalemia or very early myocardial infarction. 160 ms
The T wave inversion in V1-4 and a marked Q wave in III occur; these changes are characteristic for Ebstein's anomaly and do not reflect ischemic ECG changes in this patient. Other abnormalities that can be seen on the ECG include: signs of right atrial enlargement or tall and broad 'Himalayan' P waves
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An example of mildly elevated ST segments in V1 to V3 that are concave down An ST elevation is considered significant if the vertical distance inside the ECG trace and the baseline at a point 0.04 seconds after the J-point is at least 0.1 mV (usually representing 1 mm or 1 small square) in a limb lead or 0.2 mV (2 mm or 2 small squares) in a ...
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