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Obscured vision due to papilledema may last only seconds, while a severely atherosclerotic carotid artery may be associated with a duration of one to ten minutes. [6] Certainly, additional symptoms may be present with the amaurosis fugax, and those findings will depend on the cause of the transient monocular vision loss.
The terms low vision and blindness are often used for levels of impairment which are difficult or impossible to correct and significantly impact daily life. [7] In addition to the various permanent conditions, fleeting temporary vision impairment, amaurosis fugax, may occur, and may indicate serious medical problems. [8] [9] [10]
Amblyopia, also called lazy eye, is a disorder of sight in which the brain fails to fully process input from one eye and over time favors the other eye. [1] It results in decreased vision in an eye that typically appears normal in other aspects. [ 1 ]
Eye flashing: characterised by bursts or streaks of light that appear in an individual's field of vision. As eye flashing may indicate impending retinal detachment, medical attention is required. [8] Eyelid twitching: where the eyelid muscles contract in an irregular or abnormal pattern. Such movements can cause irritation and fatigue to the ...
Anisometropia causes some people to have mild vision problems, or occasionally more serious symptoms like alternating vision or frequent squinting. However, since most people do not show any clear symptoms, the condition usually is found during a routine eye exam. [8] For early detection in preverbal children, photoscreening can be used.
A surgical technique offered is to create a "reading eye" and a "distance vision eye", a technique commonly used in contact lens practice, known as monovision. Monovision can be created with contact lenses, so candidates for this procedure can determine if they are prepared to have their corneas reshaped by surgery to cause this effect permanently.
The term neurological visual impairment (NVI) covers both CVI and total cortical blindness. Delayed visual maturation, another form of NVI, is similar to CVI, except the child's visual difficulties resolve in a few months. Though the vision of a person with CVI may change, it rarely if ever becomes totally normal.
It is not due to a deficit in vision (acuity, visual field, and scanning), language, memory, or intellect. [1] While cortical blindness results from lesions to primary visual cortex, visual agnosia is often due to damage to more anterior cortex such as the posterior occipital and/or temporal lobe (s) in the brain. [2]
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