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  2. Aldosterone synthase - Wikipedia

    en.wikipedia.org/wiki/Aldosterone_synthase

    Aldosterone synthase, also called steroid 18-hydroxylase, corticosterone 18-monooxygenase or P450C18, is a steroid hydroxylase cytochrome P450 enzyme involved in the biosynthesis of the mineralocorticoid aldosterone and other steroids. The enzyme catalyzes sequential hydroxylations of the steroid angular methyl group at C18 after initial 11β ...

  3. Glucocorticoid remediable aldosteronism - Wikipedia

    en.wikipedia.org/wiki/Glucocorticoid_remediable...

    Glucocorticoid remediable aldosteronism. Glucocorticoid remediable aldosteronism also describable as aldosterone synthase hyperactivity, is an autosomal dominant disorder in which the increase in aldosterone secretion produced by ACTH is no longer transient. It is a cause of primary hyperaldosteronism.

  4. Familial hyperaldosteronism - Wikipedia

    en.wikipedia.org/wiki/Familial_hyperaldosteronism

    Familial hyperaldosteronism is a group of inherited conditions in which the adrenal glands, which are small glands located on top of each kidney, produce too much of the hormone aldosterone. [1] Excess aldosterone causes the kidneys to retain more salt than normal, which in turn increases the body's fluid levels and causes high blood pressure. [1]

  5. Aldosterone - Wikipedia

    en.wikipedia.org/wiki/Aldosterone

    Aldosterone is the primary of several endogenous members of the class of mineralocorticoids in humans. Deoxycorticosterone is another important member of this class. Aldosterone tends to promote Na + and water retention, and lower plasma K + concentration by the following mechanisms:

  6. Apparent mineralocorticoid excess syndrome - Wikipedia

    en.wikipedia.org/wiki/Apparent_mineralocorticoid...

    Other names. AME, 11-beta-hydroxysteroid dehydrogenase deficiency type 2, Ulick syndrome. Apparent mineralocorticoid excess syndrome has an autosomal recessive pattern of inheritance. Specialty. Medical genetics, endocrinology. Symptoms. Hypertension, hypokalemia, metabolic alkalosis, and low plasma renin activity. [1]

  7. Pseudohypoaldosteronism - Wikipedia

    en.wikipedia.org/wiki/Pseudohypoaldosteronism

    Failure to thrive, dehydration, hyponatremia, metabolic acidosis, hyperkalemia, and other non-specific symptoms including nausea, vomiting, extreme fatigue, and muscle weakness. Pseudohypoaldosteronism type 1 (PHA1) is characterized by the body's inability to respond adequately to aldosterone, a hormone crucial for regulating electrolyte levels.

  8. Primary aldosteronism - Wikipedia

    en.wikipedia.org/wiki/Primary_aldosteronism

    10% of people with high blood pressure [ 1 ] Primary aldosteronism (PA), also known as primary hyperaldosteronism, refers to the excess production of the hormone aldosterone from the adrenal glands, resulting in low renin levels and high blood pressure. [ 1 ] This abnormality is a paraneoplastic syndrome (i.e. caused by hyperplasia or tumors).

  9. Steroidogenic enzyme - Wikipedia

    en.wikipedia.org/wiki/Steroidogenic_enzyme

    Steroidogenic enzymes are enzymes that are involved in steroidogenesis and steroid biosynthesis. [2][3][4][5] They are responsible for the biosynthesis of the steroid hormones, including sex steroids (androgens, estrogens, and progestogens) and corticosteroids (glucocorticoids and mineralocorticoids), as well as neurosteroids, from cholesterol ...