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Left ventricular hypertrophy with secondary repolarization abnormalities as seen on ECG Histopathology of (a) normal myocardium and (b) myocardial hypertrophy. Scale bar indicates 50 μm. Gross pathology of left ventricular hypertrophy. Left ventricle is at right in image, serially sectioned from apex to near base.
Repolarization of the ventricle happens in the opposite direction of depolarization and is negative current, signifying the relaxation of the cardiac muscle of the ventricles. But this negative flow causes a positive T wave; although the cell becomes more negatively charged, the net effect is in the positive direction, and the ECG reports this ...
The electrical axis of the heart is the net direction in which the wave of depolarization travels. It is measured using an electrocardiogram (ECG).Normally, this begins at the sinoatrial node (SA node); from here the wave of depolarisation travels down to the apex of the heart.
Current standard ECG markers of repolarization abnormalities include ST depression, T wave inversion and QT prolongation. Many studies have investigated the prognostic strength of the SA for cardiac morbidity and mortality compared to these and other ECG parameters.
Research in the late 2000s has linked this finding to ventricular fibrillation, particularly in those who have fainted or have a family history of sudden cardiac death. [5] [6] [7] Although there is a significant relationship between ventricular fibrillation and some early repolarization's patterns, the overall lifetime occurrence of idiopathic ventricular fibrillation is exceptionally rare. [8]
Right ventricular hypertrophy (RVH) is a condition defined by an abnormal enlargement of the cardiac muscle surrounding the right ventricle.The right ventricle is one of the four chambers of the heart.
Long QT syndrome (LQTS) is a condition affecting repolarization (relaxing) of the heart after a heartbeat, giving rise to an abnormally lengthy QT interval. [7] It results in an increased risk of an irregular heartbeat which can result in fainting, drowning, seizures, or sudden death. [1]
Specifically, an increase in Q wave size, abnormalities in the P wave, as well as giant inverted T waves, are indicative of significant concentric hypertrophy. [13] Specific changes in repolarization and depolarization events are indicative of different underlying causes of hypertrophy and can assist in the appropriate management of the condition.