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The diathesis-stress model, also known as the vulnerability–stress model, is a psychological theory that attempts to explain a disorder, or its trajectory, as the result of an interaction between a predispositional vulnerability, the diathesis, and stress caused by life experiences.
Another paradigm, introduced by Zubin and Spring in 1977, was the stress-vulnerability model where the individual has unique characteristics that give him or her strengths or vulnerabilities to deal with stress, a predisposition for schizophrenia. More recently, with the decoding of the human genome, there had been a focus on identifying ...
Research has shown that schizophrenia is a polygenic disorder and that genetic vulnerability to schizophrenia is highly multifactorial, caused by the interactions of several genes with environmental risk factors. [2] Twin studies have shown that an identical twin has ~50% risk of also developing the disorder. [14]
This contrasted the popular Diathesis-Stress Model of Schizophrenia, [4] which suggested the disorder results from the interaction of predisposed individual vulnerability and external stressors. Although both models have a biopsychosocial framework, they differ in the emphases placed on each component (i.e., biological, psychological and social ...
This model of developmental instability is compatible with the diathesis–stress model. For those who argue that schizophrenia and other frontal lobe disorders are connected to delayed cerebral maturation, this pathophysiological model explains schizophrenia as one unwanted extreme of normal variation.
A related view, the diathesis-stress model, posits that mental disorders result from genetic dispositions and environmental stressors, combining to cause patterns of distress or dysfunction. [19] The model is one way to explain why some individuals are more vulnerable to mental disorders than others.
The causes of schizophrenia that underlie the development of schizophrenia, a psychiatric disorder, are complex and not clearly understood.A number of hypotheses including the dopamine hypothesis, and the glutamate hypothesis have been put forward in an attempt to explain the link between altered brain function and the symptoms and development of schizophrenia.
The model outlines how mental health problems progress to full-blown disorders when stressors exceed an individual’s coping resources and resilience. [33] In social psychiatry, the Stress-Vulnerability Model provides a framework for understanding how social and environmental factors contribute to mental illness.
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