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Pulmonary edema (British English: oedema), also known as pulmonary congestion, is excessive fluid accumulation in the tissue or air spaces (usually alveoli) of the lungs. [1] This leads to impaired gas exchange , most often leading to shortness of breath ( dyspnea ) which can progress to hypoxemia and respiratory failure .
Other causes of pulmonary edema that require rapid intervention and should be considered first include fluid overload, brain injury, and anaphylaxis. If when considering these differentials, there is no evidence for administration of excessive fluids, no focal signs suggesting a brain injury, and so signs of allergic reaction, one can then ...
However, they recommend it be considered in patients with ADHF and potentially reversible caradiogenic shock. [12] Opioids. Opioids have traditionally been used in the treatment of the acute pulmonary edema that results from acute decompensated heart failure. A 2006 review, however, found little evidence to support this practice.
The treatment of acute respiratory failure may involve medication such as bronchodilators (for airways disease), [7] [8] antibiotics (for infections), glucocorticoids (for numerous causes), diuretics (for pulmonary oedema), amongst others. [1] [9] [10] Respiratory failure resulting from an overdose of opioids may be treated with the antidote ...
Some hospitals in the U.S. are seeing an increase in RSV and higher levels of "walking pneumonia" among young children despite overall respiratory illness activity remaining low nationally.
Gaidar's aide Valery Natarov stated that Gaidar died unexpectedly, early on 16 December 2009, at his Moscow Oblast home while he was working on a book for children. Gaidar died of pulmonary edema, provoked by myocardial ischemia. [4] He is survived by his wife, three sons and daughter. [28]
A chest x-ray showing pulmonary edema with bilateral pleural effusions. Along with: Elevations in brain-natriuretic peptide (BNP) or N-terminal (NT)-pro BNP. Evidence of cardiovascular system changes (tachycardia, hypertension, widened pulse pressure, jugular venous distension, peripheral edema) Evidence of fluid overload.
In 1994, a new definition was recommended by the American-European Consensus Conference Committee [6] [10] which recognized the variability in severity of pulmonary injury. [51] The definition required the following criteria to be met: acute onset, persistent dyspnea; bilateral infiltrates on chest radiograph consistent with pulmonary edema