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Other intracellular parasites have developed different ways to enter a host cell that do not require a specific component or action from within the host cell. An example is intracellular parasites using a method called gliding motility. This is the use of an actin-myosin motor that is connected to the intracellular parasites' cytoskeleton.
Accordingly, it is convenient and customary to regard them as obligate intracellular parasites. Among the Vespidae family, Vespula austriaca is an example of an obligate reproductive parasite; its common host is Vespula acadica. [3] In the genus Bombus, B. bohemicus is an obligate parasite of B. locurum, B. cryptarum, and B. terrestris. [4]
Chlamydia trachomatis (/ k l ə ˈ m ɪ d i ə t r ə ˈ k oʊ m ə t ɪ s /) is a Gram-negative, anaerobic bacterium responsible for chlamydia and trachoma. C. trachomatis exists in two forms, an extracellular infectious elementary body (EB) and an intracellular non-infectious reticulate body (RB). [2]
As obligate intracellular parasites, they must live and reproduce within an animal cell. Coccidian parasites infect the intestinal tracts of animals, [2] and are the largest group of apicomplexan protozoa. Infection with these parasites is known as coccidiosis. Coccidia can infect all mammals, some birds, some fish, some reptiles, and some ...
Human parasites are divided into endoparasites, which cause infection inside the body, and ectoparasites, which cause infection superficially within the skin. The cysts and eggs of endoparasites may be found in feces , which aids in the detection of the parasite in the human host while also providing the means for the parasitic species to exit ...
Schistosoma mansoni is a water-borne parasite of humans, and belongs to the group of blood flukes (Schistosoma). The adult lives in the blood vessels (mesenteric veins) near the human intestine. It causes intestinal schistosomiasis (similar to S. japonicum, S. mekongi, S. guineensis, and S. intercalatum). Clinical symptoms are caused by the eggs.
The PVM surrounds the intracellular parasite, creating a separate bubble of cytoplasm-filled plasma membrane within the host cell. The rhoptry and the microneme, special secretory organelles found in apicomplexan parasites, play a major role in the formation of the vacuole. [2] [3] The PVM is extensively re-modelled by parasitic proteins. [4]
Sabin and Olitsky showed that T. gondii was an obligate intracellular parasite and that mice fed T. gondii-contaminated tissue also contracted the infection. [11] Thus Sabin and Olitsky demonstrated T. gondii as a pathogen transmissible between animals.
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