Search results
Results from the WOW.Com Content Network
Amnesia is a deficit in memory caused by brain damage or brain diseases, [1] but it can also be temporarily caused by the use of various sedative and hypnotic drugs. The memory can be either wholly or partially lost due to the extent of damage that is caused. [2] There are two main types of amnesia:
The atypical clinical syndrome of the memory disorder (as opposed to organic amnesia) is that a person with psychogenic amnesia is profoundly unable to remember personal information about themselves; there is a lack of conscious self-knowledge which affects even simple self-knowledge, such as who they are. [5]
Declarative memory consists of semantic memory and episodic memory.Semantic memory refers to acquired facts and general knowledge about the world. Examples include the name and physical attributes of objects and events, origins and history of objects, causes and effects of events or objects, associations between concepts, categories, opinions, beliefs, knowledge of historical events, etc. [2 ...
In most cases of anterograde amnesia, patients lose declarative memory, or the recollection of facts, but they retain nondeclarative memory, often called procedural memory. For instance, they are able to remember, and in some cases learn how to do things, such as talking on the phone or riding a bicycle, but they may not remember what they had ...
Korsakoff syndrome (KS) [1] is a disorder of the central nervous system characterized by amnesia, deficits in explicit memory, and confabulation.This neurological disorder is caused by a deficiency of thiamine (vitamin B 1) in the brain, and it is typically associated with and exacerbated by the prolonged, excessive ingestion of alcohol. [2]
The “spacing effect” refers to a phenomenon whereby learning, or the creation of a memory, occurs more effectively when information, or exposure to a stimulus, is spaced out.
A person experiencing a TEA episode has very little short-term memory, so that there is profound difficulty remembering events in the past few minutes (anterograde amnesia), or of events in the hours before the onset of the attack, and even memories of important events in recent years may not be accessible during the amnestic event (retrograde amnesia). [6]
They discovered that inhibiting a protein called Activin A could stop dyskinesia symptoms and essentially erase the brain’s “bad memory” response to certain Parkinson’s therapies.