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Right ventricular hypertrophy is the intermediate stage between increased right ventricular pressure (in the early stages) and right ventricle failure (in the later stages). [11] As such, management of right ventricular hypertrophy is about either preventing the development of right ventricular hypertrophy in the first place, or preventing the ...
Right atrial enlargement (RAE) is clinically significant due to its prevalence in diagnosing supraventricular arrhythmias. Further, early diagnosis using risk factors like RAE may decrease mortality because patients with RAE are at 9x more risk of arrhythmias and other cardiac conditions compared to their healthy counterparts. [ 2 ]
The underlying commonality in these disease states is an increase in pressures that the ventricles experience. For example, in tetralogy of Fallot, the right ventricle is exposed to the high pressures of the left heart due to a defect in the septum; as a result the right ventricle undergoes hypertrophy to compensate for these increased pressures.
A ventricular septal defect is a hole between the two lower chambers of the heart Tricuspid atresia. The tricuspid valve is located between the right atrium and the right ventricle.
When normal, the RV is about half the size of the left ventricle (LV). When strained, it can be as large as or larger than the LV. [5] An important potential finding with echo is McConnell's sign, where only the RV apex wall contracts; [7] it is specific for right heart strain and typically indicates a large PE. [8]
Ventricular remodeling may include ventricular hypertrophy, ventricular dilation, cardiomegaly, and other changes. It is an aspect of cardiomyopathy , of which there are many types. Concentric hypertrophy is due to pressure overload , while eccentric hypertrophy is due to volume overload .
Atrial enlargement refers to a condition where the left atrium or right atrium of the heart is larger than would be expected. It can also affect both atria. [1] Types include: Left atrial enlargement; Right atrial enlargement
In more severe cases it is a consequence of dilation of the right ventricle, leading to displacement of the papillary muscles which control the valve's ability to close. [13] Dilation of the right ventricle occurs secondary to ventricular septal defects, right to left shunting of blood, eisenmenger syndrome, hyperthyroidism, and pulmonary stenosis.
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