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Cannabis use as a medical treatment has risen globally since 2008 for a variety of reasons including increasing popular support for cannabis legalization and increased incidence of chronic pain among patients. [2] While medical cannabis use is increasing, there are major social and legal barriers which lead to cannabis research proceeding more ...
[27] [28] Long-term cannabis use may cause nausea and vomiting, a condition known as cannabinoid hyperemesis syndrome (CHS). [ 29 ] A 2016 Cochrane review said that cannabinoids were "probably effective" in treating chemotherapy-induced nausea in children, but with a high side-effect profile (mainly drowsiness, dizziness, altered moods, and ...
"abdominal pain, diarrhea, potentially carcinogenic, with others can potentiate cardiac glycosides and antiarrhythmic agents" [3] Areca nut: betel nut Areca catechu "deterioration of psychosis in patients with preexisting psychiatric disorders"; [5] known carcinogen contributing to cancer of the mouth, pharynx, esophagus and stomach when chewed ...
Delta-9-tetrahydrocannabinol, or THC, is the part of the cannabis plant that produces a “high” — a key reason marijuana helps with nausea and pain, he added.
Cannabinoid hyperemesis syndrome (CHS) is recurrent nausea, vomiting, and cramping abdominal pain that can occur due to prolonged, high-dose cannabis use. [4] [5] Complications are related to persistent vomiting and dehydration which may lead to kidney failure and electrolyte problems.
To find out, I picked the brain of Lewis Nelson, professor and chair of the department of emergency medicine and chief of the division of medical toxicology at Rutgers New Jersey Medical School.
A dried cannabis flower. The short-term effects of cannabis are caused by many chemical compounds in the cannabis plant, including 113 [clarification needed] different cannabinoids, such as tetrahydrocannabinol, and 120 terpenes, [1] which allow its drug to have various psychological and physiological effects on the human body.
One effect of 5-HT 2A receptor activation is a reduction in intraocular pressure, and so 5-HT 2A agonists can be useful for the treatment of glaucoma. This has led to the development of compounds such as AL-34662 that are hoped to reduce pressure inside the eyes but without crossing the blood–brain barrier and producing hallucinogenic side ...