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In a suspected antiphospholipid syndrome, lupus anticoagulant is generally tested in conjunction with anti-apolipoprotein antibodies and anti-cardiolipin antibodies, and diagnostic criteria require one clinical event (i.e. thrombosis or pregnancy complication) and two positive blood test results spaced at least three months apart that detect at ...
Anti-Ro/SSA antibodies are found in 40–90% of patients with systematic lupus erythematosus (SLE). The antibodies can be detected years before symptoms of SLE surface, making them an effective diagnostic tool. [3] [9] In patients with SLE, high levels of Anti-Ro/SSA are correlated with elevated levels of IFN-α.
Ninety-six percent of patients with lupus induced by procainamide will have a positive test for anti-histone antibodies, and 100% of patients whose lupus was induced by penicillamine, isoniazid, or methyldopa will have a positive test for anti-histone antibodies. In 70% of patients with rheumatoid arthritis, Felty's syndrome, Sjogren's syndrome ...
Immunological criteria: Individuals exhibiting 1) high serum levels of antinuclear antibodies; 2) high serum levels of anti-dsDNA antibodies; 3) high serum levels of anti-Sm antibodies; 4) high blood levels of antiphospholipid antibodies, high blood levels of the lupus anticoagulant, a false positive rapid plasma reagin test, or a medium to ...
Coeliac disease (antibody levels correlate with the level of intestinal damage [8] [9]), autoimmune hepatitis, gastric cancer: anti-CCP: cyclic citrullinated peptide: rheumatoid arthritis: Liver kidney microsomal type 1 antibody: autoimmune hepatitis [10] Lupus anticoagulant: Anti-thrombin antibodies: thrombin: systemic lupus erythematosus ...
Therefore, presence of anti-dsDNA antibodies are suggestive of SLE, however an absence of the antibodies does not rule out the disease. [1] The levels of circulating anti-dsDNA antibodies fluctuate with disease activity in SLE. Increases in titres of the antibodies can coincide with, or even precede an increase of disease activity.
References range may vary with age, sex, race, pregnancy, [10] diet, use of prescribed or herbal drugs and stress. Reference ranges often depend on the analytical method used, for reasons such as inaccuracy, lack of standardisation, lack of certified reference material and differing antibody reactivity. [11]
Patients with SLE have higher levels of DNA damage than normal subjects, and several proteins involved in the preservation of genomic stability show polymorphisms, some of which increase the risk for SLE development. [76] Defective DNA repair is a likely mechanism underlying lupus development. [77]
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