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  2. Hypocalcemia - Wikipedia

    en.wikipedia.org/wiki/Hypocalcemia

    The most common cause for hypocalcemia is iatrogenic hypoparathyroidism. [2] Other causes include other forms of hypoparathyroidism, vitamin D deficiency, kidney failure, pancreatitis, calcium channel blocker overdose, rhabdomyolysis, tumor lysis syndrome, and medications such as bisphosphonates or denosumab. [1]

  3. Calcium channel blocker toxicity - Wikipedia

    en.wikipedia.org/wiki/Calcium_channel_blocker...

    Calcium channel blocker toxicity is the taking of too much of the medications known as calcium channel blockers (CCBs), either by accident or on purpose. [3] This often causes a slow heart rate and low blood pressure. [1] This can progress to the heart stopping altogether. [2]

  4. Secondary hyperparathyroidism - Wikipedia

    en.wikipedia.org/wiki/Secondary_hyperparathyroidism

    If the underlying cause of the hypocalcemia can be addressed, the hyperparathyroidism will resolve. In people with chronic kidney failure, treatment consists of dietary restriction of phosphorus; supplements containing an active form of vitamin D, such as calcitriol, doxercalciferol, paricalcitol; and phosphate binders, which are either calcium-based and non-calcium based.

  5. Loop diuretic - Wikipedia

    en.wikipedia.org/wiki/Loop_diuretic

    The loss of magnesium as a result of loop diuretics has also been suggested as a possible cause of pseudogout (chondrocalcinosis). [18] Infrequent ADRs include: dyslipidemia, increased serum creatinine concentration, hypocalcemia, rash. Metabolic alkalosis may also be seen with loop diuretic use. [citation needed]

  6. Furosemide - Wikipedia

    en.wikipedia.org/wiki/Furosemide

    Other electrolyte abnormalities that can result from furosemide use include hyponatremia, hypochloremia, hypomagnesemia, and hypocalcemia. [31] In the treatment of heart failure, many studies have shown that the long-term use of furosemide can cause varying degrees of thiamine deficiency, so thiamine supplementation is also suggested. [32]

  7. Tumor lysis syndrome - Wikipedia

    en.wikipedia.org/wiki/Tumor_lysis_syndrome

    Hyperphosphatemia causes acute kidney injury in tumor lysis syndrome, because of deposition of calcium phosphate crystals in the kidney parenchyma. [2] Hypocalcemia. Because of the hyperphosphatemia, calcium is precipitated to form calcium phosphate, leading to hypocalcemia. [2] Symptoms of hypocalcemia include (but are not limited to): [9] tetany

  8. Sodium channel blocker - Wikipedia

    en.wikipedia.org/wiki/Sodium_channel_blocker

    Calcium has been shown to block sodium channels [2] which explains the effects of hypercalcemia and hypocalcemia. Lamotrigine is known to block sodium channels but it is not known whether it is extracellular or intracellular. Cannabidiol (CBD) has been shown to cause inhibitory effects on sodium currents. This voltage-dependent inhibition is ...

  9. Cinacalcet - Wikipedia

    en.wikipedia.org/wiki/Cinacalcet

    Hypocalcemia (decreased calcium levels) is a contraindication to the use of cinacalcet. Those who have serum calcium levels less than 7.5 mg/dL should not be started on cinacalcet. Hypocalcemia symptoms include paresthesias, myalgias, muscle cramping, tetany, and convulsions.