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After the pandemic hit, sales for at-home workout equipment skyrocketed. Health and fitness equipment sales doubled from March to October of 2020, reaching $2.3 billion total. With the Delta ...
Orthostatic syncope refers to syncope resulting from a postural decrease in blood pressure, termed orthostatic hypotension. [1]Orthostatic hypotension occurs when there is a persistent reduction in blood pressure of at least 20mmHg systolic or 10mmHg diastolic within three minutes of standing or being upright to 60 degrees on the head-up tilt table.
The prevalence of this condition has been studied in various populations. In a study conducted in 1922, it was found that 4.2% of 2000 apparently healthy aviators aged 18 to 42 years had an increase in diastolic blood pressure from below 90 mmHg while in the supine position to above 90 mmHg in the upright posture. [24]
A patient is considered to have orthostatic hypotension when the systolic blood pressure falls by more than 20 mm Hg, the diastolic blood pressure falls by more than 10 mm Hg, or the pulse rises by more than 20 beats per minute within 3 minutes of standing [5] [7]
The basic physiological change that occurs in the body during prolonged standing or sudden stand from supine position is that there will be increased pooling of blood in the legs. This decreases the venous return, and so there will be decreased cardiac output, which ultimately causes systolic blood pressure to fall (hypotension).
Orthostatic hypotension is defined as a fall in systolic blood pressure of at least 20 mmHg or the diastolic blood pressure of at least 10 mmHg between the supine reading and the upright reading. Also, the heart rate should be measured for both positions.
Supine hypertension is a paradoxical elevation in blood pressure upon assuming a supine position from a standing or sitting position. [1] [2] [3] It is assumed to be a manifestation of disorders of the autonomic nervous system [4] or due to side effects of medications such as midodrine and droxidopa. [5] [6]
Another experiment recently was only able to find evidence that vasodilation, not the skeletal muscle pump, was responsible for maintaining proper pressure and blood return. [7] This might have to do with the lack of rigorous physiological tests thus far used to be able to prove the pump. Experiments have shown the use of passive leg exercises ...
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