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Recurrent mild hypoglycemia may fit a reactive hypoglycemia pattern, but this is also the peak age for idiopathic postprandial syndrome, and recurrent "spells" in this age group can be traced to orthostatic hypotension or hyperventilation as often as demonstrable hypoglycemia. [citation needed] Insulin-induced hypoglycemia
Hypoglycemia was also reported due to increased secretion of insulin in the body from activation of β 2 receptors. [ citation needed ] In 2013, zilpaterol , a β agonist sold by Merck, was temporarily withdrawn due to signs of sickness in some cattle that were fed the drug.
[3] [2] Post-gastric bypass hypoglycemia can be prevented by eating smaller, more frequent meals, avoiding sugar-filled foods, as well as medical treatment with an alpha-glucosidase inhibitor, diazoxide, or octreotide. [3] [2] Some causes of hypoglycemia require treatment of the underlying cause to best prevent hypoglycemia. [2]
In insulin-deficient diabetes (exogenous) insulin levels do not decrease as glucose levels fall, and the combination of deficient glucagon and epinephrine responses causes defective glucose counterregulation. Furthermore, reduced sympathoadrenal responses can cause hypoglycemia unawareness.
Salbutamol, also known as albuterol and sold under the brand name Ventolin among others, [1] is a medication that opens up the medium and large airways in the lungs. [8] It is a short-acting β 2 adrenergic receptor agonist that causes relaxation of airway smooth muscle . [ 8 ]
It can raise the glucose by 30–100 mg/dL within minutes in any form of hypoglycemia caused by insulin excess (including all types of diabetic hypoglycemia). It comes in a glucagon emergency rescue kit which includes tiny vials containing 1 mg, which is a standard adult dose.
Reactive hypoglycemia, postprandial hypoglycemia, or sugar crash is a term describing recurrent episodes of symptomatic hypoglycemia occurring within four hours [1] after a high carbohydrate meal in people with and without diabetes. [2] The term is not necessarily a diagnosis since it requires an evaluation to determine the cause of the ...
[31] [32] These drugs are known to cause hypoglycemia and can lead to beta-cell failure due to overstimulation. [2] Second-generation versions of sulfonylureas are shorter acting and less likely to cause hypoglycemia. [32] GLP-1 receptor agonists stimulate insulin secretion by simulating activation of the body's endogenous incretin system. [32]
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