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  2. Cerebral amyloid angiopathy - Wikipedia

    en.wikipedia.org/wiki/Cerebral_amyloid_angiopathy

    Cerebral amyloid angiopathy (CAA) is a form of angiopathy in which amyloid beta peptide deposits in the walls of small to medium blood vessels of the central nervous system and meninges. [ 2 ] [ 3 ] The term congophilic is sometimes used because the presence of the abnormal aggregations of amyloid can be demonstrated by microscopic examination ...

  3. Vascular dementia - Wikipedia

    en.wikipedia.org/wiki/Vascular_dementia

    Vascular dementia can sometimes be triggered by cerebral amyloid angiopathy, which involves accumulation of amyloid beta plaques in the walls of the cerebral arteries, leading to breakdown and rupture of the vessels. [2] [5] Since amyloid plaques are a characteristic feature of Alzheimer's disease, vascular dementia may occur as a consequence ...

  4. Boston criteria - Wikipedia

    en.wikipedia.org/wiki/Boston_criteria

    The Boston criteria version 2.0 [1] is a set of guidelines designed to diagnose cerebral amyloid angiopathy (CAA), a disease that affects small blood vessels in the brain, particularly those in the cortex and leptomeninges. Although the gold standard for diagnosis is histopathological examination, the Boston criteria provide clinicians with a ...

  5. Study challenges current views about toxic plaque formation ...

    www.aol.com/study-challenges-current-views-toxic...

    Beta-amyloid, a naturally occurring brain protein, is key to the disease’s progression as it builds up in insoluble clusters, forming plaques between neurons and causing damage.

  6. Amyloid plaques - Wikipedia

    en.wikipedia.org/wiki/Amyloid_plaques

    Amyloid plaques (also known as ... onset, and progression of Alzheimer's disease. ... However, Aβ plaques (along with cerebral Aβ-amyloid angiopathy) ...

  7. Intracranial hemorrhage - Wikipedia

    en.wikipedia.org/wiki/Intracranial_hemorrhage

    Cerebral amyloid angiopathy (CAA) is the deposition of Amyloid beta peptide protein within the brain. Accumulation of such peptide proteins within the walls of the arteries can cause weakening of the walls and causes microhemorrhages, SAH within the cerebral sulci or large cerebral intraparenchymal bleed.

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