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A recent systematic review, published in 2014 [16] showed statistically significant improvement in symptoms of irritability and hyperactivity in 77% of children treated with naltrexone. Core autism symptoms were unaffected. Side effects were mild and the drug was generally well tolerated.
The review found that CBT was moderately to highly effective at reducing anxiety in school children with autism spectrum disorder, but that effects varied depending on whether they were reported by clinicians, parents or self-reported. Treatments involving parents and one-on-one compared to group treatments were more effective. [12]
Rarer side effects may indicate a dangerous allergic reaction. These include: paradoxical bronchospasm (shortness of breath and difficulty breathing); skin itching, rash, or hives ( urticaria ); swelling ( angioedema ) of any part of the face or throat (which can lead to voice hoarseness ), or swelling of the extremities.
Medications are used to reverse the symptoms of extrapyramidal side effects caused by antipsychotics or other drugs, by either directly or indirectly increasing dopaminergic neurotransmission. The treatment varies by the type of the EPS, but may involve anticholinergic agents such as procyclidine, benztropine, diphenhydramine, and trihexyphenidyl.
A study into the effects of the benzodiazepine receptor antagonist, flumazenil, on benzodiazepine withdrawal symptoms persisting after withdrawal was carried out by Lader and Morton. Study subjects had been benzodiazepine-free for between one month and five years, but all reported persisting withdrawal effects to varying degrees.
Reduced affect display, sometimes referred to as emotional blunting or emotional numbing, is a condition of reduced emotional reactivity in an individual. It manifests as a failure to express feelings either verbally or nonverbally, especially when talking about issues that would normally be expected to engage emotions.
Fragile X syndrome co-occurs with autism in many cases and is a suspected genetic cause of the autism in these cases. [11] [22] This finding has resulted in screening for FMR1 mutation to be considered mandatory in children diagnosed with autism. [11]
The cause of autism and mercury poisoning being associated is improbable because the symptoms of mercury poisoning are not present and are inherently inconsistent with the behaviors or symptoms of autism. [27] There is no accepted scientific evidence that exposure to thiomersal is a factor in causing autism. [28]
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