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Polysomnography (PSG) is a multi-parameter type of sleep study [1] and a diagnostic tool in sleep medicine.The test result is called a polysomnogram, also abbreviated PSG.The name is derived from Greek and Latin roots: the Greek πολύς (polus for "many, much", indicating many channels), the Latin somnus ("sleep"), and the Greek γράφειν (graphein, "to write").
A pulse oximeter probe applied to a person's finger. A pulse oximeter is a medical device that indirectly monitors the oxygen saturation of a patient's blood (as opposed to measuring oxygen saturation directly through a blood sample) and changes in blood volume in the skin, producing a photoplethysmogram that may be further processed into other measurements. [4]
These typically include the individual whose sleep is being studied receiving a portable monitor and may include other items such as a finger clip and an airflow sensor. Items measured include oxygen saturation, heart rate, airflow, body movement, time spent snoring, sleep position, and brain waves. [6]
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An ABG test measures the blood gas tension values of the arterial partial pressure of oxygen (PaO2), and the arterial partial pressure of carbon dioxide (PaCO2), and the blood's pH. In addition, the arterial oxygen saturation (SaO2) can be determined. Such information is vital when caring for patients with critical illnesses or respiratory disease.
The normal oxygen tension in the foot is approximately 60 mmHg, and the normal chest/foot ratio is approximately 0.9. [2] Many factors can limit the accuracy of the test including edema, temperature, inflammation, medications, and stress. In addition for the measurement to be normal, all parts of the oxygenation pathway must be functioning: the ...
Normal arterial blood oxygen saturation levels in humans are 96–100 percent. [1] If the level is below 90 percent, it is considered low and called hypoxemia . [ 2 ] Arterial blood oxygen levels below 80 percent may compromise organ function, such as the brain and heart, and should be promptly addressed.
The second is OHS primarily due to "sleep hypoventilation syndrome"; this requires a rise of CO 2 levels by 10 mmHg (1.3 kPa) after sleep compared to awake measurements and overnight drops in oxygen levels without simultaneous apnea or hypopnea. [4] [11] Overall, 90% of all people with OHS fall into the first category, and 10% in the second. [5]
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