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Pseudo-ST-depression, which is a wandering baseline due to poor skin contact of the electrode [3] Physiologic J-junctional depression with sinus tachycardia [3] Hyperventilation [3] Horizontal ST depression in V4, V5, V6 leads during a cardiac stress ECG. Other, non-ischemic, causes include: Side effect of digoxin [4] [3] Hypokalemia [4] [3]
Diagnosis is based on an ECG showing ST-segment depression at the J-point of 1 to 3 mm in leads V1 to V6, with tall and symmetrical T waves. [1] The ST-segment is upsloping and there is also often ST-segment elevation of 0.5 to 2 mm in lead aVR. [1] [2] The QRS complex is either normal or slightly wide. [1]
An ECG showing pericarditis, with ST elevation in multiple leads and slight reciprocal ST depression in aVR. Specialty: Cardiology: Symptoms: Sharp chest pain, better sitting up and worse with lying down, fever [1] Complications: Cardiac tamponade, myocarditis, constrictive pericarditis [1] [2] Usual onset: Typically sudden [1] Duration: Few ...
An upsloping, convex ST segment is highly predictive of a myocardial infarction (Pardee sign) while a concave ST elevation is less suggestive and can be found in other non-ischaemic causes. [1] Following infarction, ventricular aneurysm can develop, which leads to persistent ST elevation, loss of S wave, and T wave inversion. [1]
Typical EKG changes in acute pericarditis includes [5] [9] stage 1 -- diffuse, positive, ST elevations with reciprocal ST depression in aVR and V1. Elevation of PR segment in aVR and depression of PR in other leads especially left heart V5, V6 leads indicates atrial injury. stage 2 -- normalization of ST and PR deviations
[4] [6] Unstable angina is considered to be present in patients with ischemic symptoms suggestive of an acute coronary syndrome and no change in troponin levels, with or without changes indicative of ischemia (e.g., ST segment depression or transient elevation or new T wave inversion) on electrocardiograms. [4]
Schematic representation of normal ECG. In electrocardiography, the ST segment connects the QRS complex and the T wave and has a duration of 0.005 to 0.150 sec (5 to 150 ms). It starts at the J point (junction between the QRS complex and ST segment) and ends at the beginning of the T wave.
ECG recorded from a 17-year-old male with Lown-Ganong-Levine syndrome. LGL syndrome is diagnosed in a person who has experienced episodes of abnormal heart racing (arrhythmias) who has a PR interval less than or equal to 0.12 second (120 ms) with normal QRS complex configuration and duration on their resting ECG. [1]. [citation needed]
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