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The gastric mucosal barrier is the property of the stomach that allows it to safely contain the gastric acid required for digestion. If the barrier is broken, as by acetylsalicylic acid (ASA, aspirin) in acid solution, acid diffuses back into the mucosa where it can cause damage to the stomach itself.
An enteric coating is a polymer barrier applied to oral medication that prevents its dissolution or disintegration in the gastric environment. [1] This helps by either protecting drugs from the acidity of the stomach, the stomach from the detrimental effects of the drug, or to release the drug after the stomach (usually in the upper tract of the intestine). [2]
A gastric cytoprotectant is any medication that combats ulcers not by reducing gastric acid but by increasing mucosal protection. [citation needed] Examples of gastric cytoprotective agents include prostaglandins which protect the stomach mucosa against injury by increasing gastric mucus secretion.
Basically, stomach aches can occur whenever there is irritation of the stomach lining, says Henry Herrera, MD, a gastroenterologist at DHR Health Gastroenterology in Edinburg, Texas.
"Triple therapy" is a combination of medications to kill bacteria, reduce stomach acid production, and protect the stomach lining. [4] Antibiotics are used to treat H. pylori if it is detected. [4] Antacids are used to neutralize gastric acid but require a large dose which could lead to diarrhea. [23]
Antacids are a common treatment for mild to medium gastritis. [29] When antacids do not provide enough relief, medications such as H 2 blockers and proton-pump inhibitors that help reduce the amount of acid are often prescribed. [29] [30] Cytoprotective agents are designed to help protect the tissues that line the stomach and small intestine. [31]
The H 2 receptor antagonists are a class of drugs used to block the action of histamine on parietal cells in the stomach, decreasing the production of acid by these cells. H 2 antagonists are used in the treatment of dyspepsia, although they have been surpassed in popularity by the more effective [1] proton pump inhibitors.
Like other H 2 receptor antagonists, lafutidine acts by preventing the secretion of gastric acid. [3] It also activates calcitonin gene-related peptide, resulting in the stimulation of nitric oxide (NO) and regulation of gastric mucosal blood flow, increases somatostatin levels also resulting in less gastric acid secretion, causes the stomach lining to generate more mucin, inhibits neutrophil ...
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