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Idiopathic intracranial hypertension (IIH), previously known as pseudotumor cerebri and benign intracranial hypertension, is a condition characterized by increased intracranial pressure (pressure around the brain) without a detectable cause. [2] The main symptoms are headache, vision problems, ringing in the ears, and shoulder pain.
Intracranial hypertension (IH), also called increased ICP (IICP) or raised intracranial pressure (RICP), refers to elevated pressure in the cranium. 20–25 mmHg is the upper limit of normal at which treatment is necessary, though it is common to use 15 mmHg as the threshold for beginning treatment.
Symptoms vary depending on the location and the size of the area of involvement of the stroke. Edema, or swelling, of the brain may occur which increases intracranial pressure and may result in brain herniation. A stroke may result in coma or death if it involves key areas of the brain. [14]
Increased intracranial pressure (ICP) is a life-threatening surgical emergency marked by symptoms of headache, nausea, vomiting, decreased consciousness. [1] Symptoms are frequently accompanied by visual disturbances such as gaze paresis , reduced vision, and dizziness. [ 1 ]
Stroke-like symptoms such as confusion, weakness, and dizziness may be monitored. Headache tends to worsen over a period of several days. [ 3 ] Some of the less common symptoms include intracranial hypertension and intracranial aneurysms .
Raised intracranial pressure [1] as a result of one or more of the following: Brain tumor, idiopathic intracranial hypertension (also known as Pseudotumor Cerebri), cerebral venous sinus thrombosis or intracerebral hemorrhage; Respiratory failure [4] Isotretinoin, which is a powerful derivative of vitamin A, rarely causes papilledema.
Vertebrobasilar insufficiency (VBI) describes a temporary set of symptoms due to decreased blood flow in the posterior circulation of the brain.The posterior circulation supplies the medulla, pons, midbrain, cerebellum and (in 70-80% of people) supplies the posterior cerebellar artery to the thalamus and occipital cortex. [1]
According to this theory, dural holes and intracranial hypotension are symptoms caused by low venous pressure in the epidural space. When leg muscles pump blood towards the heart, and pressure in the inferior vena cava vein becomes negative, the network of epidural veins is overdrained, causing CSF to be aspirated into the epidural space.