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The biology of depression is the attempt to identify a biochemical origin of depression, as opposed to theories that emphasize psychological or situational causes. Scientific studies have found that different brain areas show altered activity in humans with major depressive disorder (MDD) . [ 1 ]
One of the earliest theories of addiction was the reward effect. This theory suggests that an individual consumes a substance that will elicit a pleasurable effect. The individual continues to use this substance to recreate this same feeling, ultimately becoming addicted to the sensation they receive from the substance. [34]
Models of addiction risk that have been proposed in psychology literature include an affect dysregulation model of positive and negative psychological affects, the reinforcement sensitivity theory model of impulsiveness and behavioral inhibition, and an impulsivity model of reward sensitization and impulsiveness. [1] [5] [6]
Cloninger's theory suggests that over expression of the RD temperament could cause psychiatric illnesses, such as addictive behaviors, sociopathies, and personality disorders. [ 22 ] Low levels of norepinephrine cause an increase in reward dependence.
Behavioral theories of depression emphasize the role maladaptive actions play in the onset and maintenance of depression. These theories stem from work concerning the principles of learning and conditioning from the early to mid-1900s. [3] Ivan Pavlov and B. F. Skinner are often credited with the establishment of
The neurotrophic hypothesis of depression [1] proposes that major depressive disorder (MDD) is caused, at least partly, by impaired neurotrophic support.Neurotrophic factors (also known as neurotrophins) are a family of closely related proteins which regulate the survival, development, and function of neurons in both the central and peripheral nervous systems.
While depression is a complex condition with many factors involved, it is commonly attributed to an imbalance of several key monoamine neurotransmitters, including serotonin, dopamine and norepinephrine. This monoamine hypothesis of depression is popular because of the simplicity of the explanation. [4]
The cognitive model of abnormality is one of the dominant forces in academic psychology beginning in the 1970s and its appeal is partly attributed to the way it emphasizes the evaluation of internal mental processes such as perception, attention, memory, and problem-solving. The process allows psychologists to explain the development of mental ...