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This is an accepted version of this page This is the latest accepted revision, reviewed on 10 December 2024. "Skin pigmentation" redirects here. For animal skin pigmentation, see Biological pigment. Extended Coloured family from South Africa showing some spectrum of human skin coloration Human skin color ranges from the darkest brown to the lightest hues. Differences in skin color among ...
Categorization of racial groups by reference to skin color is common in classical antiquity. [7] For example, it is found in e.g. Physiognomica, a Greek treatise dated to c. 300 BC. The transmission of the "color terminology" for race from antiquity to early anthropology in 17th century Europe took place via rabbinical literature.
Leucism (/ ˈ l uː s ɪ z əm,-k ɪ z-/) [2] [3] [4] is a wide variety of conditions that result in partial loss of pigmentation in an animal—causing white, pale, or patchy coloration of the skin, hair, feathers, scales, or cuticles, but not the eyes. [4] It is occasionally spelled leukism.
Autosomal dominant A 50/50 chance of inheritance. Sickle-cell disease is inherited in the autosomal recessive pattern. When both parents have sickle-cell trait (carrier), a child has a 25% chance of sickle-cell disease (red icon), 25% do not carry any sickle-cell alleles (blue icon), and 50% have the heterozygous (carrier) condition. [1]
The pearl cockatiels' gene does not have any visual effect on the colour pigments in the bird but instead, it affects the distribution of the colours that are already present. It actually decreases the spread of the grey family of pigments ( melanin ) and increases the spread of the yellow pigments (psittacofulvin).
The ten orbs of the Monk Skin Tone Scale. The Monk Skin Tone Scale is an open-source, 10-shade scale describing human skin color, developed by Ellis Monk in partnership with Google and released in 2023. [1]
Genodermatosis is a hereditary skin disease with three inherited modes including single gene inheritance, multiple gene inheritance and chromosome inheritance. [1] There are many different types of genodermatosis; the prevalence of genodermatosis ranges from 1 per 6000 people to 1 per 500,000 people. [2]
In 2007 one genetics lab began offering a test for what they believed was a marker for seal brown, and later for an agouti allele which they believed caused the brown color. [27] [28] However, the underlying research was never published and the test was suspended by 2015 due to unreliable results. [29] [30]