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The lymph ends its journey in the thoracic duct or right lymphatic duct, which drain into the blood circulation. [10] Several research groups have hypothesized that chronic inflammation is a key regulator in the development of lymphedema. Th cells, particularly Th2 differentiation, play a crucial role in the pathophysiology of lymphedema.
Podoconiosis causes bilateral yet asymmetrical leg swelling with overlying firm nodules. Early on, symptoms may include itching, tingling, widening of the forefoot, and swelling which then progress to soft edema, skin fibrosis, papillomatosis, and nodule formation resembling moss, giving rise to the disease's alternate name of "mossy foot" in some regions of the world. [3]
The pathophysiology is not yet well understood. Leukocytoclastic vasculitis is proposed to be the underlying cause resulting in reactive lymphedema. [2] Prolonged standing with full knee extension and minimal movement for a prolonged period of time is postulated to induce a temporary failure in pumping the venous and lymphatic systems in the calf region leading to acute gravity-dependent ...
Neil "George" Piller is an Australian professor of lymphology at the Department of Surgery, School of Medicine, Flinders University. Piller is also the Director of the Lymphoedema Assessment Unit, Flinders Surgical Oncology as well as member of the Flinders University microcirculatory and lymphological research group. [1]
Lymphedema. Lymphedema is the chronic pooling of lymph fluid in the tissue. Lymphedema can start anywhere in the lymphatic system of the body. It's also a side-effect of some surgical procedures. Kathy Bates is an advocate and supporter for further research for lymphedema. [3] Lymphocytosis. Lymphocytosis is a high lymphocyte count.
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