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In 1976, neurologist Robert Katzmann suggested a link between senile dementia and Alzheimer's disease. [283] Katzmann suggested that much of the senile dementia occurring (by definition) after the age of 65, was pathologically identical with Alzheimer's disease occurring in people under age 65 and therefore should not be treated differently. [284]
From the age of 60 years (10%) to the age of 80 years (60%), the proportion of people with senile plaques increases linearly. Women are slightly more likely to have plaques than are men. [45] [44] Both plaques and Alzheimer's disease also are more common in aging persons with trisomy-21 (Down syndrome).
The term senile dementia of the Alzheimer type (SDAT) was used for a time to describe the condition in those over 65, with classical Alzheimer's disease being used to describe those who were younger. Eventually, the term Alzheimer's disease was formally adopted in medical nomenclature to describe individuals of all ages with a characteristic ...
Pre-dementia or early-stage dementia (stages 1, 2, and 3). In this initial phase, a person can still live independently and may not exhibit obvious memory loss or have any difficulty completing ...
Compared to late onset dementia, patients with early onset dementia are more likely to have dementias other than Alzheimer's disease, although Alzheimer's is the most common etiology in either case. [13] In general, early onset dementia has a faster progression and features more extensive neurological damage when compared to late onset dementia.
In addition to this, a decrease of 20% incidence of Parkinson’s disease, as well as a 19% decrease in incidence of Alzheimer’s disease was observed in the group. Risk of vascular dementia was ...
Fischer supposedly (like Alois Alzheimer) employed new staining and autopsy results, and described "senile plaques" that are still accepted as the characteristic of the disease in addition to "neurofibrillary tangles" discovered by Alzheimer. [5] Both Fischer and Alzheimer argued that senile plaques may be formed by microorganisms. [10]
Alzheimer's disease (AD) is clinically characterized by a progressive decline in memory and cognitive functions, leading to severe dementia. Microscopically, AD is identified by the presence of two types of insoluble fibrous materials: (1) extracellular amyloid (Aβ) protein forming senile plaques and (2) intracellular neurofibrillary lesions ...