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Implants, made of synthetic materials, are naturally coated by a biofilm by the body, which may function as a favorable medium for bacteria growth. Implant failure due to bacterial infection of the implant can occur at any point of implant lifetime. Bacteria may already reside on the implant or be introduced during the implantation.
Although S. epidermidis is not usually pathogenic, patients with compromised immune systems are at risk of developing infection. These infections are generally hospital-acquired. [4] S. epidermidis is a particular concern for people with catheters or other surgical implants because it is known to form biofilms that grow on these devices. [5]
Peri-implant disease is an umbrella term for inflammatory diseases of tissues including both peri-implantitis and peri-implant mucositis. Peri-implant mucositis is a disease where inflammation is limited to the surrounding mucosa of an implant whereby peri-implantitis an inflammatory disease affecting mucosa as well as bone. [4]
According to WEAR-TV, Dr. Kolb, author of "The Naked Truth About Breast Implants," says "she's seeing lots of women with mold in their saline implants, often from defective valves."
Persister cells are highly enriched in biofilms, and this makes biofilm-related diseases difficult to treat. Examples are chronic infections of implanted medical devices such as catheters and artificial joints, urinary tract infections , middle ear infections and fatal lung disease.
The FDA rule mandating women be notified of their breast density is the result of intense lobbying by women who found champions in state and federal legislators to get laws introduced and passed.
Richards revealed during a January 3 appearance on “Jeff Lewis Live” that she had “ruptured” both of her breast implants performing one of the dangerous tasks on the reality television series.
Nitroxoline is an antibiotic [1] that has been in use in Europe for about fifty years, and has proven to be very effective at combating biofilm infections. Nitroxoline was shown to cause a decrease in the biofilm density of P. aeruginosa infections, which would allow access to the infection by the immune system in vivo. [2]