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While the acronyms are similar, reactive airway disease (RAD) and reactive airways dysfunction syndrome (RADS) are not the same. [1]Reactive airways dysfunction syndrome was first identified by Stuart M. Brooks and colleagues in 1985 as an asthma-like syndrome developing after a single exposure to high levels of an irritating vapor, fume, or smoke.
Reactive airways dysfunction syndrome (RADS) is a severe form of irritant induced asthma where respiratory symptoms usually develop in the minutes or hours after a single accidental inhalation of a high concentration of irritant gas, aerosol, vapor, or smoke. [3]
lung injury of acute onset, within 1 week of an apparent clinical insult and with the progression of respiratory symptoms; bilateral opacities on chest imaging (chest radiograph or CT) not explained by other lung pathology (e.g. effusion, lobar/lung collapse, or nodules) respiratory failure not explained by heart failure or volume overload ...
The diagnosis of lung cancer is based on chest radiograph and computed tomography (CT) scans, and is confirmed by biopsy. Biopsy is usually performed via bronchoscopy or CT-guided biopsy. Treatment and prognosis depend upon the histological type of cancer, the stage, and the patient's performance status. Treatments include surgery, chemotherapy ...
For example, if the reader thinks the x-ray being read has profusion most like the standard x-ray for category 1, but serious considered category 2 as an alternative description of the profusion, then the reading is 1/2. Close-up right lower zone 2/2 S/S Large opacities: A large opacity is defined as any opacity greater than 1 cm in diameter.
3. Nodule with poorly defined margins - Round density within the lung parenchyma, also called a tuberculoma. Nodules included in this category are those with margins that are indistinct or poorly defined (tree-in-bud sign [3]). The surrounding haziness can be either subtle or readily apparent and suggests coexisting airspace consolidation.
Squamous-cell lung carcinoma is one of the tumor types with the highest number of mutations since smoking, the main driver of the disease, is a strong mutagenic factor. [5] Inactivating mutations in lung SCC affect many tumor suppressor genes such as TP53 (mutated in 81% of cases), MLL2 (20%), CDKN2A (15%), KEAP1 (12%) and PTEN (8%).
For an exposure of 1 roentgen by gamma rays with an energy of 1 MeV, the dose in air will be 0.877 rad, the dose in water will be 0.975 rad, the dose in silicon will be 0.877 rad, and the dose in averaged human tissue will be 1 rad. [10] "rad" stands for radiation absorbed dose. [4]
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