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Hip dysplasia is an abnormality of the hip joint where the socket portion does not fully cover the ball portion, resulting in an increased risk for joint dislocation. [1] Hip dysplasia may occur at birth or develop in early life. [1] Regardless, it does not typically produce symptoms in babies less than a year old. [3]
There are typically four classes (or types) of PFFD, ranging from class A to class D, as detailed by Aitken. [4] [5]Type A — The femur bone is slightly shorter on the proximal end (near the hip), and the femoral head (the ball of the thigh bone that goes into the hip socket) may not be solid enough to be seen on X-rays at birth, but later hardens (ossifies).
There are three bones of the os coxae (hip bone) that come together to form the acetabulum. Contributing a little more than two-fifths of the structure is the ischium, which provides lower and side boundaries to the acetabulum. The ilium forms the upper boundary, providing a little less than two-fifths of the structure of the acetabulum.
The hip joint is classified as a ball and socket joint. This type of synovial joint allows for multidirectional movement and rotation. There are two bones that make up the hip joint and create an articulation between the femur and pelvis. This articulation connects the axial skeleton with the lower extremity.
X-rays of hip dysplasia are one of the two main methods of medical imaging to diagnose hip dysplasia, the other one being medical ultrasonography. [1] [2] Ultrasound imaging yields better results defining the anatomy until the cartilage is ossified. When the infant is around 3 months old a clear roentgenographic image can be achieved.
English: This diagram shows the anatomy of the ball and socket joint found in the hip. A ball and socket joint is a type of synovial joint in which the round surface of one bone fits into a round depression of another bone. 1.) The femur 2.) Femoral neck 3.) Femoral head 4) Acetabulum 5.) Acetabular Labrum 6.) Pelvis.
A delay in tooth development may also serve as an indication, whereby the absence of an adult successor slows down the normal resorption of the roots of the baby teeth, which is the progressive loss of parts of the tooth. Misplaced (ectopic) positioning of the adult teeth may be discovered upon examination or a radiograph.
The cause of the molar atrophy is thought to be enamel hypoplasia, or a deficiency in tooth enamel. The underlying dentin and pulp of the tooth is normal, but the enamel covering or molar sheath is thin and deformed, creating a smaller version of a typical tooth. [5] The grinding surface of a mulberry molar is also corrupted.