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Mineral dust airway disease is a general term used to describe complications due to inhaled mineral dust causing fibrosis and narrowing of primarily the respiratory bronchioles. [1] It is a part of a group of disorders known as pneumoconioses which is characterized by inhaled mineral dust and the effects on the lungs.
Symptoms arise 4 to 12 hours after exposure to an organic dust, and generally last from one to five days. Common generalised symptoms include fever over 38 °C, chills, myalgia and malaise. The most frequent respiratory symptoms are dyspnea and a dry cough, while a wheeze may be present less commonly.
Acute exposure to cadmium fumes may cause flu-like symptoms including chills, fever, and muscle ache sometimes referred to as "the cadmium blues." Symptoms may resolve after a week if there is no respiratory damage. More severe exposures can cause tracheobronchitis, pneumonitis, and pulmonary edema. Symptoms of inflammation may start hours ...
Occupational dust exposure occurs when small particles are generated at the workplace through the disturbance/agitation of rock/mineral, dry grain, timber, fiber, or other material. When these small particles become suspended in the air, they can pose a risk to the health of those who breath in the contaminated air.
Chlorine gas poisoning is an illness resulting from the effects of exposure to chlorine beyond the threshold limit value. Acute chlorine gas poisoning primarily affects the respiratory system , causing difficulty breathing, cough, irritation of the eyes, nose, and throat, and sometimes skin irritation .
Symptoms of dust pneumonia include high fever, chest pain, difficulty in breathing, and coughing. With dust pneumonia, dust settles all the way into the alveoli of the lungs, stopping the cilia from moving and preventing the lungs from ever clearing themselves. [citation needed] People who had dust pneumonia often died. [1]
Patients who have byssinosis have typically been exposed to cotton or just dust for an extended period of time and experience symptoms of chest tightness and coughing. [3] Patient history should reveal exposure to cotton, flax, hemp, or jute dust. Measurable change in lung function before and after working shifts is key to diagnosis.
Therapy is supportive and includes removal from further nitrogen dioxide exposure. Systemic symptoms include fever and anorexia. Electrocardiography and chest radiography can help in revealing diffuse, bilateral alveolar infiltrates. Chest radiography may be used in diagnosis and the baseline could be established with pulmonary function testing.