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Chemical formula. K 2 TeO 3 Appearance white crystals, powder Hazards GHS labelling: ... Potassium tellurite, K 2 TeO 3, is an inorganic potassium-tellurium compound. [1]
Potassium telluride is an inorganic compound with a chemical formula K 2 Te. It is formed from potassium and tellurium, making it a telluride. [2] Potassium telluride is a white powder. Like rubidium telluride and caesium telluride, it can be used as an ultraviolet detector in space.
The condition has been linked with genetic mutations in genes that code for certain ion channels that transport electrolytes (sodium and potassium) across cell membranes. The main ones are the L-type calcium channel α1-subunit [ 1 ] and potassium inward rectifier 2.6 ; [ 3 ] it is therefore classified as a channelopathy . [ 3 ]
The anode sludges contain the selenides and tellurides of the noble metals in compounds with the formula M 2 Se or M 2 Te (M = Cu, Ag, Au). At temperatures of 500 °C the anode sludges are roasted with sodium carbonate under air. The metal ions are reduced to the metals, while the telluride is converted to sodium tellurite. [38]
Tellurite is a oxyanion of tellurium with the formula TeO 2− 3. It is the ion of tellurous acid, and is chemically related to tellurium dioxide (TeO 2), whose mineral appearance also bears the name tellurite. Tellurites are typically colorless or white salts, which in some ways are comparable to sulfite. [3]
Environmentally, tellurite [TeO 3] 2-is the more abundant anion due to tellurate's [TeO 4] 2-low solubility limiting its concentration in biospheric waters. Another way to refer to the anions is tellurium's oxyanions , which happen to be relatively stable.
The condition is hypokalemic (manifests when potassium is low; not "causing hypokalemia") because a low extracellular potassium ion concentration will cause the muscle to repolarise to the resting potential more quickly, so even if calcium conductance does occur it cannot be sustained. It becomes more difficult to reach the calcium threshold at ...
Ingesting potassium can trigger attacks in affected individuals, even if blood potassium levels do not rise in response. In contrast to HyperKPP, hypokalemic periodic paralysis (noted in humans) refers to loss-of-function mutations in channels that prevent muscle depolarisation and therefore are aggravated by low potassium ion concentrations.