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Viral cardiomyopathy occurs when viral infections cause myocarditis with a resulting thickening of the myocardium and dilation of the ventricles. These viruses include Coxsackie B and adenovirus, echoviruses, influenza H1N1, Epstein–Barr virus, rubella (German measles virus), varicella (chickenpox virus), mumps, measles, parvoviruses, yellow fever, dengue fever, polio, rabies, and the ...
Pericarditis may be caused by viral, bacterial, or fungal infection. In the developing world the bacterial disease tuberculosis is a common cause, whereas in the developed world viruses are believed to be the cause of about 85% of cases. [6] Viral causes include coxsackievirus, herpesvirus, mumps virus, and HIV among others. [4]
Bacteria can also result in myocarditis, although it is rare in patients with normal heart function and without a preexisting immunodeficiency. [ 18 ] [ 21 ] A list of the most relevant infectious organisms is below.
The amount of virally infected cardiomyocytes varies in different stages of the disease. In a mouse model, at the acute stage (7 days after infection with coxsackievirus B3) approximately 10% of the myocytes are infected and could affect overall cardiac function. In chronic murine infection, the percentage of infected cardiomyocytes are much lower.
The coronavirus can damage the heart, according to a major new study which found abnormalities in the heart function of more than half of patients.
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The cause is typically a bacterial infection and less commonly a fungal infection. [1] Risk factors include valvular heart disease, including rheumatic disease, congenital heart disease, artificial valves, hemodialysis, intravenous drug use, and electronic pacemakers. [6] [7] [5] The bacteria most commonly involved are streptococci or ...
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