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Complex regional pain syndrome is uncommon, and its cause is not clearly understood. CRPS typically develops after an injury, surgery, heart attack, or stroke. [8] [12] Investigators estimate that 2–5% of those with peripheral nerve injury, [13] and 13–70% of those with hemiplegia (paralysis of one side of the body) [14] will develop CRPS.
Chronic visceral pain from persistent inflammation is pain caused by chronic inflammation of internal organs. This inflammation can be caused by noninfectious, infectious, or autoimmune mechanisms. [26] Chronic visceral pain from persistent inflammation in the head or neck region; Chronic visceral pain from persistent inflammation in the ...
Chronic systemic inflammation is the result of release of pro-inflammatory cytokines from immune-related cells and the chronic activation of the innate immune system.It can contribute to the development or progression of certain conditions such as cardiovascular disease, cancer, diabetes mellitus, chronic kidney disease, non-alcoholic fatty liver disease, autoimmune and neurodegenerative ...
It can double every 8 hours and reaches its peak at 36 to 50 hours following injury or inflammation. CRP between 100 and 500 mg/L is considered highly predictive of inflammation due to bacterial infection. Once inflammation subsides, CRP level falls quickly because of its relatively short half-life. [13]
Chronic cancer pain: pain in one of the body's organs caused by cancer damage (in internal organs, bone or skeletal muscular) is formed. Chronic pain post- traumatic or surgery : Pain that occurs 3 months after an injury or surgery, without taking into account infectious conditions and the severity of tissue damage; also, the person's past pain ...
It states, "Complex regional pain syndrome (CRPS), formerly reflex sympathetic dystrophy (RSD) or "causalgia",…" Thie "or" part is misleading in the extreme--it implies that RSD and causalgia are two names for the same thing.
SIRS is a serious condition related to systemic inflammation, organ dysfunction, and organ failure. It is a subset of cytokine storm, in which there is abnormal regulation of various cytokines. [6] SIRS is also closely related to sepsis, in which patients satisfy criteria for SIRS and have a suspected or proven infection. [2] [3] [4] [7]
This leads to the organ-specific damage typical of autoimmune disease. [19] Molecular mimicry may be particularly relevant to the tolerance breakdown linked to autoimmune neuropathies. The process known as " molecular mimicry " occurs when an infectious organism that shares epitopes from its host's afflicted tissue triggers an immune response ...