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  2. Sensory neuronopathy - Wikipedia

    en.wikipedia.org/wiki/Sensory_neuronopathy

    Sensory neuronopathy (also known as sensory ganglionopathy) is a type of peripheral neuropathy that results primarily in sensory symptoms (such as parasthesias, pain or ataxia) due to destruction of nerve cell bodies in the dorsal root ganglion. [1]

  3. Sensory ataxia - Wikipedia

    en.wikipedia.org/wiki/Sensory_ataxia

    Sensory ataxia also lacks the associated features of cerebellar ataxia such as pendular tendon reflexes, scanning dysarthria, nystagmus and broken pursuit eye movements. [citation needed] Patients with sensory ataxia often demonstrate pseudoathetosis and Romberg's sign. They usually complain of loss of balance in the dark, typically when ...

  4. Sensory ataxic neuropathy, dysarthria, and ophthalmoparesis

    en.wikipedia.org/wiki/Sensory_ataxic_neuropathy...

    This disorder is characterized by the adult-onset triad consisting of the following symptoms: sensory ataxic neuropathy, dysarthria, and ophthalmoparesis. MRIS often reveals white matter abnormalities and bilateral thalamus lesions. Other symptoms include generalized myopathy, epilepsy, and deafness. [1] [2]

  5. What to know about nervous system disease 'ataxia' - AOL

    www.aol.com/news/know-nervous-system-disease...

    As a stand-alone condition, "ataxia is a degenerative disease of the nervous system," explains Andrew Rosen, chief executive officer of the National Ataxia Foundation. There are many types ...

  6. Megavitamin-B6 syndrome - Wikipedia

    en.wikipedia.org/wiki/Megavitamin-B6_syndrome

    Megavitamin-B 6 syndrome is predominately a large fiber neuropathy characterized by sensory loss of joint position, vibration, and ataxia. [18] [26] Although it has characteristics of small fiber neuropathy in severe cases where there is impairment of pain, temperature, and autonomic functions. [62] [63] [14] [12] [59] [64] [17]

  7. Tabes dorsalis - Wikipedia

    en.wikipedia.org/wiki/Tabes_dorsalis

    Signs and symptoms may not appear for decades after the initial infection and include weakness, diminished reflexes, paresthesias (shooting and burning pains, pricking sensations, and formication), hypoesthesias (abnormally diminished sense of touch), tabetic gait (locomotor ataxia), progressive degeneration of the joints, loss of coordination, episodes of intense pain and disturbed sensation ...

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