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To date, human studies have loosely examined the behavioral characteristics of postprandial sleep, demonstrating potential shifts in EEG spectra and self-reported sleepiness. [2] To date, the only clear animal models for examining the genetic and neuronal basis for this behavior are the fruit fly, the mouse, and the nematode Caenorhabditis elegans.
Treatment of excessive daytime sleepiness (EDS) relies on identifying and treating the underlying disorder which may cure the person from the EDS. Drugs like modafinil , [ 22 ] armodafinil , [ 23 ] pitolisant [ 24 ] (Wakix), sodium oxybate (Xyrem) oral solution, have been approved as treatment for EDS symptoms in the United States.
However, fatigue during the day is a common symptom experienced by people in the later stages of dementia or even earlier with certain types of dementia, such as Lewy body dementia, due to changes ...
Chronic partial sleep deprivation is a form of sleep deprivation caused when one obtains some but inadequate sleep. Acute sleep deprivation is more widely known as the scenario in which one is awake for 24 hours or longer. [8] From student reports, 70.65% of students are sleep deprived and 50% of college students exhibit daytime sleepiness.
The problem with the assessment is that patients may only report the consequences of sleepiness: loss of energy, fatigue, weariness, difficulty remembering or concentrating, etc. It is crucial to aim for objective measures to quantify the sleepiness. A good measurement tool is the multiple sleep latency test (MSLT).
When specific treatments of the known condition do not fully suppress excessive daytime sleepiness, additional causes of hypersomnia should be sought. [14] For example, if a patient with sleep apnea is treated with CPAP ( continuous positive airway pressure ), which resolves their apneas but not their excessive daytime sleepiness, it is ...
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