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The palpebral portion acts involuntarily, closing the lids gently, as in sleep or in blinking; the orbital portion is subject to conscious control. When the entire muscle is brought into action, the skin of the forehead, temple, and cheek is drawn toward the medial angle of the orbit, and the eyelids are firmly closed, as in photophobia.
Blinking is a bodily function; it is a semi-autonomic rapid closing of the eyelid. [1] A single blink is determined by the forceful closing of the eyelid or inactivation of the levator palpebrae superioris and the activation of the palpebral portion of the orbicularis oculi, not the full open and close.
The extraocular muscles, or extrinsic ocular muscles, are the seven extrinsic muscles of the eye in humans and other animals. [1] Six of the extraocular muscles, the four recti muscles, and the superior and inferior oblique muscles, control movement of the eye. The other muscle, the levator palpebrae superioris, controls eyelid elevation.
Intrinsic ocular muscles [1] or intraocular muscles [2] are muscles of the inside of the eye structure. The intraocular muscles are responsible for the protraction and retraction of the eyelids and reaction to light and pupil accommodation . [ 2 ]
The zygomatic branches of the facial nerve (malar branches) are nerves of the face.They run across the zygomatic bone to the lateral angle of the orbit.Here, they supply the orbicularis oculi muscle, and join with filaments from the lacrimal nerve and the zygomaticofacial branch of the maxillary nerve (CN V 2).
Eyelid edema is a condition in which the eyelids are swollen and tissues contain excess fluid. It may affect eye function when it increases the intraocular pressure. Eyelid edema is caused by allergy, trichiasis or infections. [4] The main symptoms are swollen red eyelids, pain, and itching. Chronic eyelid edema can lead to blepharochalasis.
The lateral palpebral raphe is formed from the lateral ends of the orbicularis oculi muscle. [1] [2] It may also be formed from the pretarsal muscles of the eyelids. [3] [4] It is attached to the margin of the frontosphenoidal process of the zygomatic bone. It passes towards the midline to the lateral commissure of the eyelids.
The main first-line therapy is periodic injections of botulinum toxin type A to induce localized, partial paralysis of the eyelid muscles. [ 46 ] [ 47 ] Injections are generally administered at intervals of around 10 weeks, with variations based on patient response and usually give fairly quick relief from the muscle spasms.