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In vitro studies indicate that vitamin D increases the expression of the tyrosine hydroxylase gene in adrenal medullary cells and affects the synthesis of neurotrophic factors, nitric oxide synthase, and glutathione, which may control the body's response and adaption to stress. [20] VDR expression decreases with age. [1]
The proanthocyanidins found in cranberries are responsible for fighting this bacteria. While more research is needed, Manaker cites one study that found drinking ½ cup of proanthocyanidin-rich ...
Vitamin D and Omega-3 Trial (VITAL) [1] was a clinical trial designed to investigate the use of daily dietary supplements of vitamin D and fish oil.. The sponsor of the study was Brigham and Women's Hospital, collaborating with The National Cancer Institute, National Heart, Lung, and Blood Institute, Office of Dietary Supplements, National Institute of Neurological Disorders and Stroke ...
Giovannucci is known for his research into the health benefits of obtaining vitamin D through sunlight exposure. [4] [5] He has said that for every death caused by skin cancer resulting from sun exposure, 30 deaths might be prevented due to increased vitamin D intake, [4] and that "It does seem that Vitamin D levels seem to be a real predictor of heart disease."
Vitamin D, a potent immunomodulator, has also been shown to suppress Th17 cell differentiation and function by several research groups. [84] The active form of vitamin D has been found to 'severely impair' [85] production of the IL17 and IL-17F cytokines by Th17 cells.
Also a 2008 study published in Cancer Research has shown the addition of vitamin D 3 (along with calcium) to the diet of some mice fed a regimen similar in nutritional content to a new Western diet with 1000 IU cholecalciferol per day prevented colon cancer development. [33]
1α-Hydroxyvitamin D 5 is a chemical derivative of vitamin D 5.The motive to study 1α-hydroxyvitamin D 5 as a potential pharmaceutical drug stemmed from the tendency of calcitriol, a natural metabolite produced in the kidney, to cause toxic hypercalcemia in patients when dosed at concentrations needed to interrupt prostate cancer cells' cycle and stimulate apoptosis.
Norman's research was in the area of cellular and molecular endocrinology, where he was internationally known for his breakthroughs in the study of vitamin D. [2] This included the mechanisms of action of the steroid hormone 1,25-dihydroxycholecalciferol, vitamin D structure-function relationships, and actions of the vitamin D receptor (VDR).
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