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  2. Hypokalemia - Wikipedia

    en.wikipedia.org/wiki/Hypokalemia

    Hypokalemia is a low level of potassium (K +) in the blood serum. [1] Mild low potassium does not typically cause symptoms. [3] Symptoms may include feeling tired, leg cramps, weakness, and constipation. [1]

  3. Hyperkalemia - Wikipedia

    en.wikipedia.org/wiki/Hyperkalemia

    In sweat glands potassium elimination is quite similar to the kidney; its excretion is also controlled by aldosterone. [medical citation needed] Regulation of serum potassium is a function of intake, appropriate distribution between intracellular and extracellular compartments, and effective bodily excretion.

  4. Hypokalemic periodic paralysis - Wikipedia

    en.wikipedia.org/wiki/Hypokalemic_periodic_paralysis

    Recovery is usually sudden when it occurs, due to release of potassium from swollen muscles as they recover. Some patients may fall into an abortive attack or develop chronic muscle weakness later in life. Some people only develop symptoms of periodic paralysis due to hyperthyroidism (overactive thyroid).

  5. The Top 6 Health Benefits That Make Cucumbers So Good ... - AOL

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  6. Here’s Why Women May Want to Eat More Potassium-Rich ... - AOL

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  7. Cucumber juice - Wikipedia

    en.wikipedia.org/wiki/Cucumber_juice

    Cucumbers are 98% water. [2] Cucumber juice is used in beverages such as cocktails like the Bloody Mary, [3] dishes such as cucumber soup, [4] and in dips and salad dressings, such as green goddess dressing. [5] Cucumber juice has significant amounts of potassium [1] and is high in vitamin A. [6] It also contains sterol [7] and significant ...

  8. Berberine can upset your gastrointestinal system, causing symptoms like: nausea, diarrhea, constipation, gas and abdominal pain, Simmons says. It’s also possible that taking berberine can ...

  9. Hyperaldosteronism - Wikipedia

    en.wikipedia.org/wiki/Hyperaldosteronism

    Secondary hyperaldosteronism (also hyperreninism, or hyperreninemic hyperaldosteronism) is due to overactivity of the renin–angiotensin–aldosterone system (RAAS).. The causes of secondary hyperaldosteronism are accessory renal veins, fibromuscular dysplasia, reninoma, renal tubular acidosis, nutcracker syndrome, ectopic tumors, massive ascites, left ventricular failure, and cor pulmonale.