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[7] [72] Initially, post-stroke individuals have flaccid paralysis. [70] As recovery begins, and progresses, basic movement synergies will develop into more complex and difficult movement combinations. [7] [72] Concurrently, spasticity may develop and become quite severe before it begins to decline (if it does at all).
Ophthalmoparesis refers to weakness (-paresis) or paralysis (-plegia) of one or more extraocular muscles which are responsible for eye movements. It is a physical finding in certain neurologic, ophthalmologic, and endocrine disease. Internal ophthalmoplegia means involvement limited to the pupillary sphincter and ciliary muscle.
Oculomotor nerve palsy or oculomotor neuropathy [1] is an eye condition resulting from damage to the third cranial nerve or a branch thereof. As the name suggests, the oculomotor nerve supplies the majority of the muscles controlling eye movements (four of the six extraocular muscles, excluding only the lateral rectus and superior oblique).
Superior alternating hemiplegia (also known as Weber syndrome) has a few distinct symptoms: contralateral hemiparesis of limb and facial muscle accompanied by weakness in one or more muscles that control eye movement on the same side. [2] Another symptom that appears is the loss of eye movement due to damage to the oculomotor nerve fibers.
One pupil is dilated and unreactive, while the other is normal (in this case, the right eye is dilated, while the left eye is normal in size). This could mean damage to the oculomotor nerve (cranial nerve number 3, CN III) on the right side, or indicate the possibility of vascular involvement. Both pupils are dilated and unreactive to light.
In this photo provided by UPMC and Pitt Health Sciences, neurosurgeon Dr. Peter Gerszten tells study participant Heather Rendulic how electrodes will be implanted on her spinal cord at UPMC ...
In children, the most common cause is a stroke of the ventral pons. [9]Unlike persistent vegetative state, in which the upper portions of the brain are damaged and the lower portions are spared, locked-in syndrome is essentially the opposite, caused by damage to specific portions of the lower brain and brainstem, with no damage to the upper brain.
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