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  2. Adenosine monophosphate deaminase deficiency type 1

    en.wikipedia.org/wiki/Adenosine_monophosphate...

    Adenosine monophosphate deaminase deficiency type 1 or AMPD1, is a human metabolic disorder in which the body consistently lacks the enzyme AMP deaminase, [1] in sufficient quantities. This may result in exercise intolerance, muscle pain and muscle cramping. The disease was formerly known as myoadenylate deaminase deficiency (MADD).

  3. Hypophosphatemia - Wikipedia

    en.wikipedia.org/wiki/Hypophosphatemia

    Hypophosphatemia is an electrolyte disorder in which there is a low level of phosphate in the blood. [1] Symptoms may include weakness, trouble breathing, and loss of appetite. [1]

  4. Metabolic myopathy - Wikipedia

    en.wikipedia.org/wiki/Metabolic_myopathy

    Some people with a metabolic myopathy never develop symptoms due to the body's ability to produce enough ATP through alternative pathways (e.g. the majority of those with AMP-deaminase deficiency are asymptomatic [1] [21]). H 2 O + ATP → H + + ADP + P i + energy → muscle contraction [22] ATP is needed for muscle contraction by two processes:

  5. Mitochondrial myopathy - Wikipedia

    en.wikipedia.org/wiki/Mitochondrial_myopathy

    Mitochondrial myopathies are types of myopathies associated with mitochondrial disease. [1] Adenosine triphosphate (), the chemical used to provide energy for the cell, cannot be produced sufficiently by oxidative phosphorylation when the mitochondrion is either damaged or missing necessary enzymes or transport proteins.

  6. Pyruvate kinase deficiency - Wikipedia

    en.wikipedia.org/wiki/Pyruvate_kinase_deficiency

    With these severe cases of pyruvate kinase deficiency in red blood cells, treatment is the only option, there is no cure. However, treatment is usually effective in reducing the severity of the symptoms. [12] [17] The most common treatment is blood transfusions, especially in infants and young children.

  7. Hereditary fructose intolerance - Wikipedia

    en.wikipedia.org/wiki/Hereditary_fructose...

    HFI is caused by a deficiency of aldolase B. [5] A deficiency of aldolase B results in an accumulation of fructose-1-phosphate, and trapping of phosphate (fructokinase requires adenosine triphosphate (ATP)). The downstream effects of this enzyme block are the inhibition of glucose production and reduced regeneration of ATP. [5]

  8. MT-ATP6 - Wikipedia

    en.wikipedia.org/wiki/MT-ATP6

    Mitochondrial complex V deficiency is a shortage (deficiency) or loss of function in complex V of the electron transport chain that can cause a wide variety of signs and symptoms affecting many organs and systems of the body, particularly the nervous system and the heart. The disorder can be life-threatening in infancy or early childhood.

  9. Mitochondrial disease - Wikipedia

    en.wikipedia.org/wiki/Mitochondrial_disease

    Mitochondrial disease is a group of disorders caused by mitochondrial dysfunction.Mitochondria are the organelles that generate energy for the cell and are found in every cell of the human body except red blood cells.